@article{mukherjee_unipolar_1992,
	title = {Unipolar mania reconsidered-a {CT} scan study},
	volume = {31},
	journal = {Biol Psychiatry},
	author = {S. Mukherjee and D. B. Schnur and R. Reddy},
	year = {1992},
	pages = {248}
},

@article{venkataraman_mania_1992,
	title = {Mania associated with fluoxetine treatment in adolescents},
	volume = {31},
	journal = {J Am Acad Child Adolesc Psychiatry},
	author = {S. Venkataraman and M. W. Naylor and C. A. King},
	year = {1992},
	pages = {276--281}
},

@article{gustafsson_serotonin_2006,
	title = {Serotonin and fluoxetine modulate bone cell function in vitro},
	volume = {98},
	number = {1},
	journal = {Journal of cellular biochemistry},
	author = {B. I. Gustafsson and L. Thommesen and A. K. Stunes and K. Tommeras and I. Westbroek and H. L. Waldum and K. Slordahl and M. V. Tamburstuen and J. E. Reseland and U. Syversen},
	year = {2006}
},

@article{sakkas_induction_2003,
	title = {Induction of mania by {rTMS:} report of two cases},
	volume = {18},
	number = {4},
	journal = {European Psychiatry},
	author = {P. Sakkas and P. Mihalopoulou and P. Mourtzouhou and C. Psarros and V. Masdrakis and A. Politis and G. N. Christodoulou},
	year = {2003},
	pages = {196--198}
},

@article{azab_mechanisms_2008,
	title = {Mechanisms of action of the mood stabilizer valproate: a focus on {GSK-3} inhibition},
	volume = {3},
	number = {4},
	journal = {Future Neurol.},
	author = {A. N. Azab and J. F. Ishak and J. Kaplanski and V. Delbar and M. L. Greenberg},
	year = {2008},
	pages = {433--445}
},

@article{barkin_induction_1997,
	title = {Induction of mania by risperidone resistant to mood stabilizers.},
	volume = {17},
	number = {1},
	journal = {Journal of clinical psychopharmacology},
	author = {J. S. Barkin and V. M. Pais Jr and M. F. Gaffney},
	year = {1997},
	pages = {57}
},

@article{barbenel_mania_2000,
	title = {Mania in a patient receiving testosterone replacement post-orchidectomy taking St John's wort and sertraline},
	volume = {14},
	number = {1},
	journal = {Journal of Psychopharmacology},
	author = {D. Barbenel and B. Yusufi and D. {O'Shea} and C. Bench},
	year = {2000},
	pages = {84}
},

@article{lyras_chronic_2002,
	title = {Chronic high dose {L-DOPA} alone or in combination with the {COMT} inhibitor entacapone does not increase oxidative damage or impair the function of the nigro-striatal pathway in normal cynomologus monkeys},
	volume = {109},
	url = {http://dx.doi.org/10.1007/s702-002-8236-2},
	doi = {10.1007/s702-002-8236-2},
	abstract = {Summary.   Parkinson's disease {(PD)} is characterised by a loss of pigmented dopaminergic neurones in the zona compacta of substantia
nigra. The mechanisms underlying nigral cell death remain unknown but may involve oxidative damage. There has been concern
that {L-DOPA} treatment may accelerate nigral pathology in {PD} through chemical and enzymatic oxidation to reactive oxygen species.
In the present study, we examined tissues from normal macaque monkeys treated for 13 weeks with high doses of {L-DOPA} (in combination
with the peripheral decarboxylase inhibitor, carbidopa) and/or the {COMT} inhibitor, entacapone. Plasma was analysed for changes
in pro-tein carbonyls as a marker of oxidative damage to protein. Cortical tissue was examined for changes in levels of protein
carbonyls, lipid peroxidation and oxidative damage to {DNA.} The integrity of the nigro-striatal pathway was assessed by nigral
tyrosine hydroxylase {mRNA} levels and specific {[3H]mazindol} binding to dopaminergic terminals in caudate-putamen.

No alterations in plasma protein carbonyls were observed in any treatment group. An increase was found in the levels of protein
carbonyls, lipid peroxidation and {5-OH} uracil, but not other products of oxidative {DNA} damage, in cerebral cortex of monkeys
treated with {L-DOPA} plus carbidopa or with {L-DOPA} plus carbidopa and entacapone but this was only statistically significant
in the latter group. There was no change in nigral tyrosine hydroxylase {mRNA} levels or specific striatal {[3H]mazindol} binding in brain tissue from monkeys treated with either {L-DOPA} plus carbidopa or {L-DOPA} plus carbidopa and entacapone.
The results show that in the normal monkeys {L-DOPA} does not provoke marked oxidative damage even at high doses, and that there
is little or no potentiation of its effects by entacapone. Neither {L-DOPA} plus carbidopa nor {L-DOPA} plus carbidopa and entacapone
led to obvious damage to the nigro-striatal pathway.


},
	number = {1},
	journal = {Journal of Neural Transmission},
	author = {L. Lyras and {B.-Y.} Zeng and G. {McKenzie} and R. K. B. Pearce and B. Halliwell and P. Jenner},
	year = {2002},
	pages = {53--67}
},

@article{morishita_induction_2003,
	title = {Induction of mania in depression by paroxetine},
	volume = {18},
	number = {7},
	journal = {Human Psychopharmacology: Clinical and Experimental},
	author = {S. Morishita and S. Arita},
	year = {2003}
},

@book{margolese_hypomania_2003,
	title = {Hypomania induced by adjunctive lamotrigine},
	volume = {160},
	publisher = {Am Psychiatric Assoc},
	author = {{HC} {MARGOLESE} and L. {BEAUCLAIR} and N. {SZKRUMELAK} and {GUY} {CHOUINARD}},
	year = {2003},
	pages = {183--184}
},

@article{rendell_risperidone_????,
	title = {Risperidone alone or in combination for acute mania {(Review)}},
	author = {J. M. Rendell and H. J. Gijsman and M. S. Bauer and G. M. Goodwin and J. R. Geddes}
},

@article{berthier_fluoxetine--induced_1993,
	title = {Fluoxetine--induced mania in a patient with post-stroke depression},
	volume = {163},
	number = {5},
	journal = {The British Journal of Psychiatry},
	author = {M. L. Berthier and J. Kulisevsky},
	year = {1993},
	pages = {698b}
},

@article{teieher_fluoxetine_????,
	title = {Fluoxetine Side Effects},
	volume = {147},
	journal = {Am. J. Psychiatry},
	author = {M. H. Teieher and ? Glod and J. O. Cole and W. M. Grecnberg},
	pages = {370--371}
},

@article{konitsiotis_ampa_2000,
	title = {{AMPA} receptor blockade improves levodopa-induced dyskinesia in {MPTP} monkeys},
	volume = {54},
	url = {http://www.neurology.org/cgi/content/abstract/54/8/1589},
	abstract = {{OBJECTIVE:} To evaluate the contribution of amino-3-hydroxy-5-methyl-4-isoxazole proprionic acid {(AMPA)} glutamate receptors to the pathogenesis of parkinsonian signs and levodopa-induced dyskinesias. {BACKGROUND:} Motor fluctuations and dyskinesias reflect, in part, altered function of glutamate receptors of the {NMDA} subtype. The possible role of {AMPA} receptors, however, has not yet been examined. {METHODS:} The authors compared the ability of an {AMPA} agonist {(CX516)} and a noncompetitive {AMPA} antagonist {(LY300164)} to alter parkinsonian symptoms and levodopa-induced dyskinesia in {MPTP-lesioned} monkeys. Eight levodopa-treated parkinsonian monkeys received rising doses of each drug, first in monotherapy and then in combination with low-, medium-, and high-dose levodopa. {RESULTS:} {CX516} alone, as well as when combined with low-dose levodopa, did not affect motor activity but induced dyskinesia. Moreover, following injection of the higher doses of levodopa, it increased levodopa-induced dyskinesia by up to 52\% (p {\textless} 0.05). {LY300164} potentiated the motor activating effects of low-dose levodopa, increasing motor activity by as much as 86\% (p {\textless} 0.05), and that of medium-dose levodopa as much as 54\% (p {\textless} 0.05). At the same time, {LY300164} decreased levodopa-induced dyskinesia by up to 40\% (p {\textless} 0.05). {CONCLUSIONS:} {AMPA} receptor upregulation may contribute to the expression of levodopa-induced dyskinesia. Conceivably, noncompetitive {AMPA} receptor antagonists could be useful, alone or in combination with {NMDA} antagonists, in the treatment of {PD,} by enhancing the antiparkinsonian effects of levodopa without increasing and possibly even decreasing levodopa-induced dyskinesia.
},
	number = {8},
	journal = {Neurology},
	author = {S. Konitsiotis and P. J. Blanchet and L. Verhagen and E. Lamers and T. N. Chase},
	month = apr,
	year = {2000},
	pages = {1589--1595}
},

@article{lewis_induction_1982,
	title = {The induction of mania. A natural history study with controls.},
	volume = {39},
	number = {3},
	journal = {Archives of general psychiatry},
	author = {J. L. Lewis and G. Winokur},
	year = {1982},
	pages = {303}
},

@article{foland_evidence_2008,
	title = {Evidence for deficient modulation of amygdala response by prefrontal cortex in bipolar mania},
	volume = {162},
	issn = {0925-4927},
	url = {http://www.sciencedirect.com/science/article/B6TBW-4RB5BH6-2/2/f00d105ee9b5cdfc727e73647d4f78da},
	doi = {10.1016/j.pscychresns.2007.04.007},
	abstract = {
Several studies have implicated the involvement of two major components of emotion regulatory networks, the ventrolateral prefrontal cortex {(VLPFC)} and amygdala, in the pathophysiology of bipolar disorder. In healthy subjects, the {VLPFC} has been shown to negatively modulate amygdala response when subjects cognitively evaluate an emotional face by identifying and labeling the emotion it expresses. The current study used such a paradigm to assess whether the strength of this modulation was altered in bipolar subjects when manic. During functional magnetic resonance imaging {(fMRI),} nine manic subjects with bipolar I disorder and nine healthy subjects either named the emotion shown in a face by identifying one of two words that correctly expressed the emotion (emotion labeling task) or matched the emotion shown in a face to one of two other faces (emotion perception task). The degree to which the {VLPFC} regulated amygdala response during these tasks was assessed using a psychophysiological interaction {(PPI)} analysis. Compared with healthy subjects, manic patients had a significantly reduced {VLPFC} regulation of amygdala response during the emotion labeling task. These findings, taken in context with previous {fMRI} studies of bipolar mania, suggest that reductions in inhibitory frontal activity in these patients may lead to an increased reactivity of the amygdala.},
	number = {1},
	journal = {Psychiatry Research: Neuroimaging},
	author = {Lara C. Foland and Lori L. Altshuler and Susan Y. Bookheimer and Naomi Eisenberger and Jennifer Townsend and Paul M. Thompson},
	year = {2008},
	keywords = {Bipolar {disorder,Emotion} {regulation,fMRI,Functional} connectivity},
	pages = {27--37}
},

@book{kinrys_hypomania_2000,
	title = {Hypomania Associated With ?3 Fatty Acids},
	volume = {57},
	publisher = {Am Med Assoc},
	author = {G. Kinrys},
	year = {2000},
	pages = {715--716}
},

@article{sternbach_fluoxetine-associated_1991,
	title = {{Fluoxetine-Associated} Potentiation of {Calcium-Channel} Blockers.},
	volume = {11},
	number = {6},
	journal = {Journal of Clinical Psychopharmacology},
	author = {H. {STERNBACH}},
	year = {1991},
	pages = {390}
},

@article{short_hypomania_1995,
	title = {Hypomania induced by gabapentin},
	volume = {166},
	number = {5},
	journal = {The British Journal of Psychiatry},
	author = {C. Short and L. Cooke},
	year = {1995},
	pages = {679b}
},

@article{horrigan_guanfacine_1999,
	title = {Guanfacine and secondary mania in children},
	volume = {54},
	number = {3},
	journal = {Journal of affective disorders},
	author = {J. P. Horrigan and L. Jarrett Barnhill},
	year = {1999},
	pages = {309--314}
},

@article{strakowski_functional_2004,
	title = {The functional neuroanatomy of bipolar disorder: a review of neuroimaging findings},
	volume = {10},
	number = {1},
	journal = {Molecular Psychiatry},
	author = {S. M. Strakowski and M. P. {DelBello} and C. M. Adler},
	year = {2004},
	pages = {105--116}
},

@article{pfohl_unipolar_1982,
	title = {Unipolar vs. bipolar mania: a review of 247 patients},
	volume = {141},
	number = {5},
	journal = {The British Journal of Psychiatry},
	author = {B. Pfohl},
	year = {1982},
	pages = {453--458}
},

@article{saba_catatonia_2002,
	title = {Catatonia and Transcranial Magnetic Stimulation},
	volume = {159},
	url = {http://ajp.psychiatryonline.org},
	doi = {10.1176/appi.ajp.159.10.1794},
	number = {10},
	journal = {Am J Psychiatry},
	author = {{GHASSEN} {SABA} and {JEAN} {FRANCOIS} {ROCAMORA} and {KHALID} {KALALOU} and {RENE} {BENADHIRA} and {MARION} {PLAZE} and {BEATRICE} {AUBRIOT-DELMAS} and {DOMINIQUE} {JANUEL}},
	month = oct,
	year = {2002},
	pages = {1794}
},

@article{hcrsh_transient_????,
	title = {Transient psychosis with fluoxetine},
	volume = {30},
	journal = {Psychiatry},
	author = {C. B. Hcrsh and M. S. Sokol and C. R. Pfeffer and ? Nakra and P. Szwabo and G. Grossberg},
	pages = {179--186}
},

@article{semba_effect_1988,
	title = {Effect of monoamine precursors on the forced-swimming test in mice},
	volume = {95},
	url = {http://dx.doi.org/10.1007/BF00174513},
	doi = {{10.1007/BF00174513}},
	abstract = {The antidepressant properties of monoamine precursors were evaluated by the forced-swimming test for mice developed by Porsolt et al. {DOPA} but not 5-hydroxy-tryptophan {(5HTP)} shortened immobility at doses that did not increase locomotor activity. Although l-threo-dihydroxyphenylserine {(DOPS),} an artificial norepinephrine {(NE)} precursor, did not change immobility in intact mice, {DOPS} significantly reduced immobility in mice pretreated with the selective {NE} neurotoxin {DSP4.} These results suggest possible antidepressant properties of {DOPA} and {DOPS,} the latter of which may act as an antidepressant in a certain {NE-depleting} condition.},
	number = {2},
	journal = {Psychopharmacology},
	author = {Jun-ichi Semba and Ryo Takahashi},
	month = jun,
	year = {1988},
	pages = {222--225}
},

@article{godbout_laboratory_1998,
	title = {A Laboratory Study of Sleep and Dreaming in a Case of Asperger's Syndrome},
	volume = {8},
	url = {http://dx.doi.org/10.1023/B:DREM.0000005898.95212.58},
	doi = {{10.1023/B:DREM.0000005898.95212.58}},
	abstract = {Asperger's Syndrome {(AS)} is a pervasive developmental disorder whose continuity with {High-Functioning} Autism is still a matter of debate. Clinical observations suggest that patients with {AS} may present the same sleep disorders as autistic patients, including difficulties in initiating and maintaining sleep as well as poor dream recall. We recorded the sleep of a 25-year-old male patient with {AS} for two nights using a full {EEG} montage and compared the second night to that of a group of normal participants. We found low levels of slow wave sleep {(SWS:} stages 3 + 4), high levels of stage 1, and a large number of awakenings. The organization of {REM} sleep was unremarkable, including normal {REM} density. Analyses of phasic {EEG} events revealed a very low incidence of sleep spindles and a normal number of K-complexes over bilateral frontal and central {EEG} leads. In order to collect dream reports, the patient was awakened three times over two nights following at least 15 minutes of {REM} sleep in each case. On each occasion the patient was not aware of any mental activity happening just prior to awakening. These observations are discussed with regards to the connections that may exist between {EEG} sleep spindle activity, selective attention, and the capacity to generate a dream report.},
	number = {2},
	journal = {Dreaming},
	author = {Roger Godbout and Cybèle Bergeron and Emmanuel Stip and Laurent Mottron},
	month = jun,
	year = {1998},
	pages = {75--88}
},

@article{reddy_yc_alprazolam-induced_1996,
	title = {Alprazolam-induced hypomania},
	volume = {30},
	number = {4},
	journal = {Australian and New Zealand journal of psychiatry},
	author = {J. {REDDY} {YC} and S. Khanna and U. Anand and A. Banerjee},
	year = {1996},
	pages = {550--552}
},

@article{arana_alprazolam-induced_1985,
	title = {Alprazolam-induced mania: two clinical cases},
	volume = {142},
	number = {3},
	journal = {American Journal of Psychiatry},
	author = {G. W. Arana and C. Pearlman and R. I. Shader},
	year = {1985},
	pages = {368}
},

@article{kessler_recognition_????,
	title = {The Recognition and Management of Mania},
	author = {R. C. Kessler and K. A. {McGonagle} and S. Zhao}
},

@article{settle_jr_case_1984,
	title = {A case of mania associated with fluoxetine},
	volume = {141},
	number = {2},
	journal = {American Journal of Psychiatry},
	author = {E. C. Settle Jr and G. P. Settle},
	year = {1984}
},

@article{brown_aripiprazole_????,
	title = {Aripiprazole alone or in combination for acute mania {(Protocol)}},
	author = {R. N. Brown and M. J. Taylor and H. Wilder and J. Geddes}
},

@article{ruiz_[high-dose_????,
	title = {{[High-dose} fluoxetine-induced mania. Review and case report]},
	volume = {13},
	number = {48},
	journal = {Vertex {(Buenos} Aires, Argentina)},
	author = {M. Ruiz and C. Vairo and D. Matusevich and C. Finkelsztein},
	pages = {93}
},

@article{moses_st._2000,
	title = {St. John's Wort: Three Cases of Possible Mania Induction.},
	volume = {20},
	number = {1},
	journal = {Journal of Clinical Psychopharmacology},
	author = {E. L. Moses and A. G. Mallinger},
	year = {2000},
	pages = {115}
},

@article{savitz_dissociative_2004,
	title = {Dissociative Identity Disorder Associated with Mania and Change in Handedness.},
	volume = {17},
	number = {4},
	journal = {Cognitive and Behavioral Neurology},
	author = {J. Savitz and M. Solms and E. Pietersen and R. Ramesar and P. {Flor-Henry}},
	year = {2004},
	pages = {233}
},

@article{ne_buspirone-induced_1988,
	title = {Buspirone-induced hypomania: a case report},
	volume = {8},
	journal = {J Clin Psychopharmacol},
	author = {L. Ne and V. K. Yeragani},
	year = {1988},
	pages = {226--227}
},

@article{nakra_mania_1989,
	title = {Mania induced by fluoxetine},
	volume = {146},
	number = {11},
	journal = {American Journal of Psychiatry},
	author = {B. R. Nakra and P. Szwabo and G. T. Grossberg},
	year = {1989},
	pages = {1515}
},

@article{budman_persistent_1991,
	title = {Persistent dyskinesia in a patient receiving fluoxetine},
	volume = {148},
	number = {10},
	journal = {American Journal of Psychiatry},
	author = {C. L. Budman and R. D. Bruun},
	year = {1991},
	pages = {1403}
},

@article{landry_withdrawal_1997,
	title = {Withdrawal Hypomania Associated With Paroxetine.},
	volume = {17},
	number = {1},
	journal = {Journal of clinical psychopharmacology},
	author = {P. Landry and L. Roy},
	year = {1997},
	pages = {60}
},

@misc{d_l_murphy_mental_2003,
	type = {review-article},
	title = {Mental Effects of {L-Dopa}},
	url = {http://arjournals.annualreviews.org/doi/abs/10.1146/annurev.me.24.020173.001233},
	author = {D L Murphy},
	month = nov,
	year = {2003},
	howpublished = {http://arjournals.annualreviews.org/doi/abs/10.1146/annurev.me.24.020173.001233}
},

@article{ngr_abnormal_2008,
	title = {Abnormal Glutamatergic Neurotransmission and {Neuronal-Glial} Interactions in Acute Mania},
	volume = {64},
	issn = {0006-3223},
	url = {http://www.sciencedirect.com/science/article/B6T4S-4SXRTS7-1/2/273b8d64e3f77c05637841c8c1bba28d},
	doi = {10.1016/j.biopsych.2008.05.014},
	abstract = {Background
At excitatory synapses, glutamate released from neurons is taken up by glial cells and converted to glutamine, which is cycled back to neurons. Alterations in this system are believed to play a role in the pathophysiology of bipolar disorder, but they have not been characterized in vivo. We examined the glutamine/glutamate ratio and levels of other metabolites in acute mania and schizophrenia in this exploratory {study.Methods}
Data were obtained from 2 × 2 × 2 cm voxels in the anterior cingulate cortex {(ACC)} and parieto-occipital cortex {(POC)} using two-dimensional J-resolved proton magnetic resonance spectroscopy at 4 Tesla and analyzed using {LCModel.} Fifteen bipolar disorder patients with acute mania and 17 schizophrenia patients with acute psychosis were recruited from an inpatient unit; 21 matched healthy control subjects were also studied. Glutamine/glutamate ratio and N-acetylaspartate, creatine, choline, and myo-inositol levels were evaluated in a repeated measures design. Medication effects and relationship to demographic and clinical variables were {analyzed.Results}
Glutamine/glutamate ratio was significantly higher in {ACC} and {POC} in bipolar disorder, but not schizophrenia, compared with healthy control subjects. N-acetylaspartate was significantly lower in the {ACC} in schizophrenia. Patients on and off lithium, anticonvulsants, or benzodiazepines had similar glutamine/glutamate {ratios.Conclusions}
The elevated glutamine/glutamate ratio is consistent with glutamatergic overactivity and/or defective neuronal-glial coupling in acute mania, although medication effects cannot be ruled out. Abnormalities in glutamatergic neurotransmission and glial cell function in bipolar disorder may represent targets for novel therapeutic interventions.},
	number = {8},
	journal = {Biological Psychiatry},
	author = {Dost Öngür and J. Eric Jensen and Andrew P. Prescot and Caitlin Stork and Miriam Lundy and Bruce M. Cohen and Perry F. Renshaw},
	month = oct,
	year = {2008},
	keywords = {Bipolar {disorder,glia,glutamate,glutamine,MRS,schizophrenia}},
	pages = {718--726}
},

@article{brown_ziprasidone_????,
	title = {Ziprasidone alone or in combination for acute mania {(Protocol)}},
	author = {R. N. Brown and J. R. Geddes and S. Stockton}
},

@article{murphy_reduced_1972,
	title = {Reduced Monoamine Oxidase Activity in Blood Platelets from Bipolar Depressed Patients},
	volume = {128},
	url = {http://ajp.psychiatryonline.org/cgi/content/abstract/128/11/1351},
	doi = {10.1176/appi.ajp.128.11.1351},
	abstract = {Cellular monoamine oxidase {(MAO)} activity measured in blood platelets is significantly (45 percent) lower in depressed patients with a history of mania (bipolar patients) than in nonbipolar depressed patients and normal controls of similar age and sex. This difference in {MAO} activity may be related to other biological and psychological characteristics of these patients that suggest that bipolar patients constitute a specific subgroup among patients with affective disorders.
},
	number = {11},
	journal = {Am J Psychiatry},
	author = {{DENNIS} L. {MURPHY} and {RICHARD} {WEISS}},
	month = may,
	year = {1972},
	pages = {1351--1357}
},

@article{wernicke_safety_2004,
	title = {Safety and side effect profile of fluoxetine},
	volume = {3},
	number = {5},
	journal = {eds},
	author = {J. F. Wernicke},
	year = {2004},
	pages = {495--504}
},

@article{damore_medication-induced_1998,
	title = {Medication-induced hypomania in Asperger's disorder},
	volume = {37},
	number = {3},
	journal = {Journal of the American Academy of Child \& Adolescent Psychiatry},
	author = {J. Damore and J. Stine and L. Brody},
	year = {1998},
	pages = {248--249}
},

@article{ferre_l-dopa_1987,
	title = {L-dopa causes an acute, partial and reversible reversal of denervation-induced supersensitivity of striatal dopaminergic receptors},
	volume = {91},
	url = {http://dx.doi.org/10.1007/BF00217074},
	doi = {{10.1007/BF00217074}},
	abstract = {Abstract  Denervation-induced supersensitivity of the striatal dopamine receptors can be quantified by the turning behaviour induced by apomorphine. With this experimental model we found that high-dose L-dopa/carbidopa administration reduced this supersensitivity. This effect was seen on the 1st day and did not alter over 5 or 10 days of treatment, but dissappeared when medication was discontinued. The degree of reduction was the same, independent of the dose and period of administration. This effect could provide a useful model for studying the phenomenon of the irreversibility of the supersensitivity of the striatal dopamine receptors.},
	number = {2},
	journal = {Psychopharmacology},
	author = {S. Ferre and M. Casas and A. Cobos and C. Garcia and F. Jane and J. Grau},
	month = feb,
	year = {1987},
	pages = {254--256}
},

@book{michael_fluoxetine-induced_2000,
	title = {Fluoxetine-induced anaesthesia of vagina and nipples},
	volume = {176},
	publisher = {{RCP}},
	author = {A. Michael and C. Mayer},
	year = {2000},
	pages = {299--299}
},

@article{bahrami_brain_????,
	title = {Brain complexity increases in mania},
	author = {B. Bahrami and R. Seyedsadjadi and B. Babadi and M. Noroozian}
},

@article{savitz_dissociative_2004-1,
	title = {Dissociative identity disorder associated with mania and change in handedness},
	volume = {17},
	issn = {1543-3633},
	url = {http://www.ncbi.nlm.nih.gov/pubmed/15622020},
	abstract = {{OBJECTIVE:} To investigate the overlap between dissociative and bipolar disorders with reference to their neurophysiological foundations. {BACKGROUND:} Case reports of anomalous lateralization and shifts in handedness associated with both affective and dissociative conditions have intermittently surfaced in the literature. The two disorders are, however, usually considered to be distinct psychopathological entities. {METHOD:} A case of co-occurring bipolar disorder and dissociative identity disorder {(DID)} is presented. {RESULTS:} The "switch" in personality coincided with manic or hypomanic symptoms and was associated with a change in handedness. {CONCLUSIONS:} A parallel between the "personality" shifts that characterize {DID} and the mood fluctuations that underlie bipolar disorder is drawn, suggesting some nosological overlap between the two disorders. The possibility that these two psychiatric conditions share a similar neurophysiological architecture is also raised.},
	number = {4},
	journal = {Cognitive and Behavioral Neurology: Official Journal of the Society for Behavioral and Cognitive Neurology},
	author = {Jonathan Savitz and Mark Solms and Elize Pietersen and Rajkumar Ramesar and Pierre {Flor-Henry}},
	month = dec,
	year = {2004},
	note = {{PMID:} 15622020},
	keywords = {{Adolescent,Adult,Bipolar} {Disorder,Female,Functional} {Laterality,Multiple} Personality {Disorder,Neuropsychological} {Tests,Personality}},
	pages = {233--7}
},

@article{wilcox_abuse_1987,
	title = {Abuse of fluoxetine by a patient with anorexia nervosa},
	volume = {144},
	number = {8},
	journal = {American Journal of Psychiatry},
	author = {J. A. Wilcox},
	year = {1987},
	pages = {1100}
},

@article{bornke_acute_1998,
	title = {Acute mania due to a right hemisphere infarction},
	volume = {5},
	number = {4},
	journal = {{THE} {OFFICIAL} {JOURNAL} {OF} {THE} {EUROPEAN} {FEDERATION} {OF} {NEUROLOGICAL} {SOCIETIES}},
	author = {C. Bornke and T. Postert and H. Przuntek and T. Buttner},
	year = {1998},
	pages = {407--409}
},

@article{rabkin_adverse_1985,
	title = {Adverse Reactions to Monoamine Oxidase Inhibitors. Part {II.} Treatment Correlates and Clinical Management.},
	volume = {5},
	number = {1},
	journal = {Journal of Clinical Psychopharmacology},
	author = {J. G. Rabkin and F. M. Quitkin and P. {McGrath} and W. Harrison and E. Tricamo},
	year = {1985},
	pages = {2}
},

@article{utters_transient_????,
	title = {Transient Psychosis with Fluoxetine},
	volume = {1},
	journal = {Child and Adolescent},
	author = {I. {UTTERS}},
	pages = {193--198}
},

@article{reddy_yc_alprazolam-induced_1996-1,
	title = {Alprazolam-induced hypomania},
	volume = {30},
	number = {4},
	journal = {Australian and New Zealand journal of psychiatry},
	author = {J. {REDDY} {YC} and S. Khanna and U. Anand and A. Banerjee},
	year = {1996},
	pages = {550--552}
},

@article{altshuler_blunted_2005,
	title = {Blunted Activation in Orbitofrontal Cortex During Mania: A Functional Magnetic Resonance Imaging Study},
	volume = {58},
	issn = {0006-3223},
	url = {http://www.sciencedirect.com/science/article/B6T4S-4HMX9BN-6/2/72ef8da8114c5ae20d24386b4a9dafb2},
	doi = {10.1016/j.biopsych.2005.09.012},
	abstract = {Background
Patients with bipolar disorder have been reported to have abnormal cortical function during mania. In this study, we sought to investigate neural activity in the frontal lobe during mania, using functional magnetic resonance imaging {(fMRI).} Specifically, we sought to evaluate activation in the lateral orbitofrontal cortex, a brain region that is normally activated during activities that require response {inhibition.Methods}
Eleven manic subjects and 13 control subjects underwent {fMRI} while performing the {Go-NoGo} task, a neuropsychological paradigm known to activate the orbitofrontal cortex in normal subjects. Patterns of whole-brain activation during {fMRI} scanning were determined with statistical parametric mapping. Contrasts were made for each subject for the {NoGo} minus Go conditions. Contrasts were used in a second-level analysis with subject as a random {factor.Results}
Functional {MRI} data revealed robust activation of the right orbitofrontal cortex {(Brodmann's} area {[BA]} 47) in control subjects but not in manic subjects. Random-effects analyses demonstrated significantly less magnitude in signal intensity in the right lateral orbitofrontal cortex {(BA} 47), right hippocampus, and left cingulate {(BA} 24) in manic compared with control {subjects.Conclusions}
Mania is associated with a significant attenuation of task-related activation of right lateral orbitofrontal function. This lack of activation of a brain region that is usually involved in suppression of responses might account for some of the disinhibition seen in mania. In addition, hippocampal and cingulate activation seem to be decreased. The relationship between this reduced function and the symptoms of mania remain to be further explored.},
	number = {10},
	journal = {Biological Psychiatry},
	author = {Lori L. Altshuler and Susan Y. Bookheimer and Jennifer Townsend and Manuel A. Proenza and Naomi Eisenberger and Fred Sabb and Jim Mintz and Mark S. Cohen},
	month = nov,
	year = {2005},
	keywords = {Functional magnetic resonance imaging,mania,orbitofrontal cortex},
	pages = {763--769}
},

@article{bauer_adverse_1996,
	title = {Adverse events and tolerability of the combination of fluoxetine/lithium compared with fluoxetine.},
	volume = {16},
	number = {2},
	journal = {Journal of clinical psychopharmacology},
	author = {M. Bauer and M. Linden and B. Schaaf and H. J. Weber},
	year = {1996},
	pages = {130}
},

@article{pande_light-induced_1985,
	title = {Light-induced hypomania},
	volume = {142},
	number = {9},
	journal = {American Journal of Psychiatry},
	author = {A. C. Pande},
	year = {1985},
	pages = {1126}
},

@article{pickar_implications_1978,
	title = {Implications of monoamine oxidase inhibitor-induced hypomania},
	volume = {35},
	journal = {Arch Gen Psychiatry},
	author = {D. Pickar and R. Cohen},
	year = {1978},
	pages = {1393--1394}
},

@article{peet_induction_1994,
	title = {Induction of mania with selective serotonin re-uptake inhibitors and tricyclic antidepressants},
	volume = {164},
	number = {4},
	journal = {The British Journal of Psychiatry},
	author = {M. Peet},
	year = {1994},
	pages = {549--550}
},

@article{mackay_comparison_1997,
	title = {A comparison of fluvoxamine, fluoxetine, sertraline and paroxetine examined by observational cohort studies},
	volume = {6},
	number = {4},
	journal = {Pharmacoepidemiology and Drug Safety},
	author = {F. J. Mackay and N. R. Dunn and L. V. Wilton and G. L. Pearce and S. N. Freemantle and R. D. Mann},
	year = {1997}
},

@article{maneeton_fluoxetine_????,
	title = {Fluoxetine plus insight meditation therapy for an {SLE} pa-tient with depressive disorder: a two-year follow-up},
	author = {B. {MANEETON} and S. {MARTIN} and N. {MANEETON}}
},

@article{strakowski_mri_2008,
	title = {{MRI} brain activation in first-episode bipolar mania during a response inhibition task},
	volume = {2},
	url = {http://www.pubmedcentral.nih.gov/articlerender.fcgi?artid=2613305},
	doi = {10.1111/j.1751-7893.2008.00082.x},
	number = {4},
	journal = {Early intervention in psychiatry},
	author = {Stephen M. Strakowski and Caleb M. Adler and Michael A. Cerullo and James C. Eliassen and Martine Lamy and David E. Fleck and {Jing-Huei} Lee and Melissa P. {DelBello}},
	month = nov,
	year = {2008},
	note = {{PMC2613305}},
	pages = {225–233}
},

@article{hoffmann_identical_1987,
	title = {Identical twins' nonidentical responses to lithium},
	volume = {144},
	number = {9},
	journal = {American Journal of Psychiatry},
	author = {W. F. Hoffmann},
	year = {1987},
	pages = {1240}
},

@article{silverstone_bipolar_2005,
	title = {Bipolar disorder and {\textless}i{\textgreater}myo{\textless}/i{\textgreater}-inositol: a review of the magnetic resonance spectroscopy findings},
	volume = {7},
	url = {http://dx.doi.org/10.1111/j.1399-5618.2004.00174.x},
	doi = {10.1111/j.1399-5618.2004.00174.x},
	abstract = {Silverstone {PH,} {McGrath} {BM,} Kim H. Bipolar disorder and myo-inositol: a review of the magnetic resonance spectroscopy findings. Bipolar Disord 2005: 7: 1201310. � Blackwell Munksgaard, {2005Objectives:�} Myo-inositol is an important component of the phosphatidylinositol second messenger system {(PI-cycle).} Alterations in {PI-cycle} activity have been suggested to be involved in the pathophysiology and/or treatment of bipolar disorder. More specifically, lithium has been suggested to act primarily by lowering myo-inositol concentrations, the so-called inositol-depletion hypothesis. {myo-Inositol} concentrations can be measured in vivo with magnetic resonance spectroscopy {(MRS).Methods:�} The current review primarily examines animal and human {MRS} studies that evaluated the role of myo-inositol in bipolar illness and {treatment.Results:�} Studies have been carried out in patients who are manic, depressed, and euthymic, both on and off treatment. However, there are several limitations of these {studies.Conclusions:�} The preclinical and clinical {MRS} findings were generally supportive of the involvement of myo-inositol in bipolar disorder and its treatment. Overall, in bipolar patients who are manic or depressed there are abnormalities in brain myo-inositol concentrations, with changes in frontal and temporal lobes, as well as the cingulate gyrus and basal ganglia. These abnormalities are not seen in either euthymic patients or healthy controls, possibly due to a normalizing effect of treatment with either lithium or sodium valproate. There is also increasing evidence that sodium valproate may also act upon the {PI-cycle.} Nonetheless, it remains uncertain if these changes in myo-inositol concentration are primary or secondary. Findings regarding the specific inositol-depletion hypothesis are also generally supportive in acutely ill patients, although it is not yet possible to definitively confirm or refute this hypothesis based on the current {MRS} evidence.},
	number = {1},
	journal = {Bipolar Disorders},
	author = {Peter H Silverstone and Brent M {McGrath} and Hyeonjin Kim},
	year = {2005},
	pages = {1--10}
},

@article{janardhan_alprazolam-induced_1996,
	title = {Alprazolam-induced hypomania},
	volume = {30},
	number = {4},
	journal = {Australian and New Zealand Journal of Psychiatry},
	author = {R. Y. C. Janardhan and S. Khanna and U. Anand and A. Banerjee},
	year = {1996},
	pages = {550--552}
},

@article{mcivor_buspirone-induced_1991,
	title = {Buspirone-induced mania},
	volume = {158},
	number = {1},
	journal = {The British Journal of Psychiatry},
	author = {R. {McIvor} and K. Sinanan},
	year = {1991},
	pages = {136}
},

@article{spinella_hypomania_2002,
	title = {Hypomania induced by herbal and pharmaceutical psychotropic medicines following mild traumatic brain injury},
	volume = {16},
	number = {4},
	journal = {Brain Injury},
	author = {M. Spinella and L. A. Eaton},
	year = {2002},
	pages = {359--367}
},

@article{steiner_fluoxetine-induced_1991,
	title = {Fluoxetine-induced mania in a patient with obsessive-compulsive disorder},
	volume = {148},
	number = {10},
	journal = {American Journal of Psychiatry},
	author = {W. Steiner},
	year = {1991},
	pages = {1403}
},

@article{steiner_medication-induced_????,
	title = {{Medication-Induced} Hypomania in Asperger's Disorder},
	author = {H. Steiner and J. Lock}
},

@article{frey_proton_2005,
	title = {A proton magnetic resonance spectroscopy investigation of the dorsolateral prefrontal cortex in acute mania},
	volume = {20},
	url = {http://dx.doi.org/10.1002/hup.671},
	doi = {10.1002/hup.671},
	abstract = {Several neurochemical abnormalities have been reported in bipolar disorder {(BD),} but the exact mechanisms that underlie its pathophysiology remain to be elucidated. Proton magnetic resonance spectroscopy {(1HMRS)} allows in vivo measurements of certain neurometabolites in the human brain. {1HMRS} was used to investigate the dorsolateral prefrontal cortex {(DLPFC)} in bipolar subjects during a manic or mixed phase. {N-acetyl-L-aspartate} {(NAA),} choline-containing molecules {(Cho),} creatine plus phosphocreatine {(Cr)} and myoinositol {(Ino)} were {measured.Ten} bipolar patients (nine manic, one mixed), diagnosed by a semi-structured clinical interview {(SCID),} and ten age- and gender-matched healthy volunteers were studied. Absolute neurometabolites levels were measured from two 8�cm3 voxels placed in left and right {DLPFC} using a short {TE} {1HMRS} method at {1.5�T.} T1- and T2-weighted anatomical magnetic resonance imaging was performed to exclude any neuroanatomical {abnormality.No} significant differences were found for {NAA,} Cho, Cr, Ino, {NAA/Cr,} {Cho/Cr} or {Ino/Cr} between patients and controls. Manic/mixed patients had significantly higher left-to-right myoinositol ratios in {DLPFC} {(p�=�0.044).Increased} left-to-right myoinositol ratios in the {DLPFC} in bipolar patients during acute mania may represent a dysfunction in the phosphoinositide-signaling pathway. Longitudinal studies with larger samples of unmedicated patients assessing pre- and post-treatment times will be required for further clarification of the time course of these abnormalities and the relationship with treatment effects. Copyright � 2005 John Wiley \& Sons, Ltd.},
	number = {2},
	journal = {Human Psychopharmacology: Clinical and Experimental},
	author = {Ben�cio N. Frey and Marcelo Folgierini and Mark Nicoletti and Rodrigo {Machado-Vieira} and Jeffrey A. Stanley and Jair C. Soares and Fl�vio Kapczinski},
	year = {2005},
	pages = {133--139}
},

@article{perlis_serotonin_2003,
	title = {Serotonin transporter polymorphisms and adverse effects with fluoxetine treatment},
	volume = {54},
	number = {9},
	journal = {Biological Psychiatry},
	author = {R. H. Perlis and D. Mischoulon and J. W. Smoller and Y. J. Y. Wan and S. {Lamon-Fava} and K. M. Lin and J. F. Rosenbaum and M. Fava},
	year = {2003},
	pages = {879--883}
},

@article{amsterdam_treatment_1996,
	title = {Treatment algorithms in treatment-resistant depression},
	volume = {19},
	number = {2},
	journal = {Psychiatric Clinics of North America},
	author = {J. D. Amsterdam and M. {Hornig-Rohan}},
	year = {1996},
	pages = {371--386}
},

@article{vieta_predictors_????,
	title = {Predictors of switching from mania to depression in a large observational study across Europe {(EMBLEM)}},
	volume = {In Press, Corrected Proof},
	issn = {0165-0327},
	url = {http://www.sciencedirect.com/science/article/B6T2X-4VT0GWN-1/2/a2264a80b36e3892f474d869fe7f6aae},
	doi = {10.1016/j.jad.2009.02.007},
	abstract = {Background
The risk of switching from mania to depression in bipolar disorder has been poorly studied. Large observational studies may be useful in identifying variables that predict switch to depression after mania and provide data on medication use and outcomes in "real world" {patients.Method}
{EMBLEM} {(European} Mania in Bipolar Longitudinal Evaluation of Medication) is a 2-year, prospective, observational study of patients with a manic/mixed episode. Symptom severity measures included Clinical Global {Impression-Bipolar} Disorder scale {(CGI-BP),} Young Mania Rating Scale {(YMRS)} and 5-item Hamilton Depression Rating Scale. Switching was defined using {CGI-BP} mania and depression such that patients changed from manic and not depressed to depressed but not manic over two consecutive observations within the first 12 weeks of follow-up. Cox proportional hazards models identified baseline variables independently associated with switch to {depression.Results}
Of 2390 patients who participated in the maintenance phase (i.e. up to 24 months), 120 (5.0\%) switched to depression within the first 12 weeks. Factors associated with greater switching to depression include previous depressive episodes, substance abuse, greater {CGI-BP} overall severity and benzodiazepine use. Factors associated with lower switching rates were greater {CGI-BP} depression, lower {YMRS} severity and atypical antipsychotic {use.Limitations}
The definition of switching biased against patients with mixed episodes being likely to {switch.Conclusions}
Strictly defined, switch to depression from mania occurs in a small proportion of bipolar patients. Clinical history, illness severity, co-morbidities and treatment patterns are associated with switching to depression. Atypical antipsychotics may protect against switch to depression.},
	journal = {Journal of Affective Disorders},
	author = {Eduard Vieta and Jules Angst and Catherine Reed and Jordan Bertsch and Josep Maria Haro},
	keywords = {Bipolar {disorder,Depression,Mania,Prospective} observational {study,Switching}}
},

@article{frye_increased_2007,
	title = {Increased anterior cingulate/medial prefrontal cortical glutamate and creatine in bipolar depression},
	volume = {32},
	number = {12},
	journal = {Neuropsychopharmacology},
	author = {M. A. Frye and J. Watzl and S. Banakar and J. {O'Neill} and J. Mintz and P. Davanzo and J. Fischer and J. W. Chirichigno and J. Ventura and S. Elman},
	year = {2007},
	pages = {2490--2499}
},

@article{lloyd_epidemiology_2002,
	title = {The epidemiology of first-onset mania},
	journal = {Textbook in Psychiatric Epidemiology},
	author = {T. Lloyd and P. B. Jones},
	year = {2002},
	pages = {445–458}
},

@article{shabani_mood-stabilizing_2007,
	title = {{Mood-Stabilizing} Effect of {Twice-Weekly} Administration of Fluoxetine in a Bipolar {II} Disorder Patient},
	volume = {9},
	number = {5},
	journal = {Primary Care Companion to The Journal of Clinical Psychiatry},
	author = {A. Shabani},
	year = {2007},
	pages = {392}
},

@article{liegghio_buspirone-induced_1988,
	title = {{Buspirone-Induced} Hypomania: A Case Report.},
	volume = {8},
	number = {3},
	journal = {Journal of Clinical Psychopharmacology},
	author = {N. E. Liegghio and V. K. Yeragani},
	year = {1988},
	pages = {226}
},

@article{murphy_l-dopa_1973,
	title = {{L-Dopa,} Dopamine, and Hypomania},
	volume = {130},
	url = {http://ajp.psychiatryonline.org/cgi/content/abstract/130/1/79},
	doi = {10.1176/appi.ajp.130.1.79},
	abstract = {Greater urinary excretion of dopamine during L-dopa administration was observed in bipolar than in unipolar depressed patients. The authors suggest that increased levels of brain dopamine may play a role in the development of hypomania and mania, since there is a tendency for bipolar patients to regularly develop hypomanic episodes during L-dopa treatment and since evidence from animal studies indicates that increased brain dopamine is highly correlated with increased psychomotor activity.
},
	number = {1},
	journal = {Am J Psychiatry},
	author = {{DENNIS} L. {MURPHY} and {FREDERICK} K. {GOODWIN} and H. {KEITH} H. {BRODIE} and {WILLIAM} E. {BUNNEY}},
	year = {1973},
	pages = {79--82}
},

@article{steiner_fluoxetine-induced_1991-1,
	title = {Fluoxetine-induced mania in a patient with obsessive-compulsive disorder},
	volume = {148},
	number = {10},
	journal = {American Journal of Psychiatry},
	author = {W. Steiner},
	year = {1991},
	pages = {1403}
},

@article{xu_eight-to-ten-year_????,
	title = {An eight-to-ten-year outcome study of unipolar mania},
	volume = {90},
	journal = {Arch Psychiatry. 1992; 4 (1): 88},
	author = {W. Y. Xu and J. Z. Chen}
},

@article{spinella_hypomania_2002-1,
	title = {Hypomania induced by herbal and pharmaceutical psychotropic medicines following mild traumatic brain injury},
	volume = {16},
	number = {4},
	journal = {Brain Injury},
	author = {M. Spinella and L. A. Eaton},
	year = {2002},
	pages = {359--367}
},

@article{lavin_adverse_1993,
	title = {Adverse Reaction to {High-Dose} Fluoxetine.},
	volume = {13},
	number = {6},
	journal = {Journal of clinical psychopharmacology},
	author = {M. R. Lavin and A. Mendelowitz and S. H. Block},
	year = {1993},
	pages = {452}
},

@article{freinhar_androgen-induced_1985,
	title = {Androgen-induced hypomania.},
	volume = {46},
	number = {8},
	journal = {The Journal of clinical psychiatry},
	author = {J. P. Freinhar and W. Alvarez},
	year = {1985},
	pages = {354}
},

@article{vieta_reboxetine-induced_2001,
	title = {Reboxetine-induced hypomania.},
	volume = {62},
	number = {8},
	journal = {J Clin Psychiatry},
	author = {E. Vieta and F. Colom and A. {Martinez-Aran} and M. Reinares and A. Benabarre and B. Corbella and C. Gasto},
	year = {2001},
	pages = {655--6}
},

@article{koehler_acute_1978,
	title = {Acute confabulatory psychosis: A rare form of unipolar mania?},
	volume = {57},
	number = {5},
	journal = {Acta Psychiatrica Scandinavica},
	author = {K. Koehler and C. Jacoby},
	year = {1978},
	pages = {415--425}
},

@article{kurlan_pilot_1993,
	title = {A Pilot Controlled Study of Fluoxetine for {Obsessive-Compulsive} Symptoms in Children with Tourette's Syndrome.},
	volume = {16},
	number = {2},
	journal = {Clinical Neuropharmacology},
	author = {R. Kurlan and P. G. Como and C. Deeley and M. {McDermott} and M. P. {McDermott}},
	year = {1993},
	pages = {167}
},

@article{settle_jr_case_1984-1,
	title = {A case of mania associated with fluoxetine},
	volume = {141},
	number = {2},
	journal = {American Journal of Psychiatry},
	author = {E. C. Settle Jr and G. P. Settle},
	year = {1984}
},

@article{bachmann_mood_2005,
	title = {Mood stabilizers target cellular plasticity and resilience cascades},
	volume = {32},
	number = {2},
	journal = {Molecular neurobiology},
	author = {R. F. Bachmann and R. J. Schloesser and T. D. Gould and H. K. Manji},
	year = {2005},
	pages = {173--202}
},

@article{rachid_possible_2004,
	title = {Possible induction of mania or hypomania by atypical antipsychotics: an updated review of reported cases.},
	volume = {65},
	number = {11},
	journal = {The Journal of clinical psychiatry},
	author = {F. Rachid and G. Bertschy and G. Bondolfi and J. M. Aubry},
	year = {2004},
	pages = {1537}
},

@article{yatham_neurochemical_2003,
	title = {Neurochemical brain imaging studies in bipolar disorder.},
	volume = {15},
	number = {6},
	journal = {Acta Neuropsychiatrica},
	author = {L. N. Yatham and G. S. Malhi},
	year = {2003},
	pages = {381}
},

@article{jerome_hypomania_1991,
	title = {Hypomania with fluoxetine.},
	volume = {30},
	number = {5},
	journal = {J Am Acad Child Adolesc Psychiatry},
	author = {L. Jerome},
	year = {1991},
	pages = {850--1}
},

@article{galli_adolescent_1998,
	title = {An adolescent male with multiple paraphilias successfully treated with fluoxetine},
	volume = {8},
	number = {3},
	journal = {Journal of Child and Adolescent Psychopharmacology},
	author = {{VB} {GALLI} and {NJ} {RAUTE} and B. J. {McCONVILLE} and S. L. {McELROY}},
	year = {1998},
	pages = {195--197}
},

@article{benazzi_mania_1998,
	title = {Mania induced by olanzapine},
	volume = {13},
	number = {8},
	journal = {Human Psychopharmacology: Clinical and Experimental},
	author = {F. Benazzi and E. Rossi},
	year = {1998}
},

@article{koek_probable_1996,
	title = {Probable induction of mania by risperidone.},
	volume = {57},
	number = {4},
	journal = {The Journal of clinical psychiatry},
	author = {R. J. Koek and C. C. Kessler},
	year = {1996},
	pages = {174}
},

@article{wehr_sleep-loss_1991,
	title = {Sleep-loss as a possible mediator of diverse causes of mania},
	volume = {159},
	number = {4},
	journal = {The British Journal of Psychiatry},
	author = {T. A. Wehr},
	year = {1991},
	pages = {576--578}
},

@article{strakowski_characterizing_2009,
	title = {Characterizing Impulsivity in Mania},
	volume = {11},
	url = {http://www.pubmedcentral.nih.gov/articlerender.fcgi?artid=2626636},
	doi = {10.1111/j.1399-5618.2008.00658.x},
	number = {1},
	journal = {Bipolar disorders},
	author = {Stephen M. Strakowski and David E. Fleck and Melissa P. {DelBello} and Caleb M. Adler and Paula K. Shear and Susan L. {McElroy} and Paul E. Keck, Jr and Quinton Moss and Michael A. Cerullo and Renu Kotwal and Stephan Arndt},
	month = feb,
	year = {2009},
	note = {{PMC2626636}},
	pages = {41–51}
},

@article{kumar_sertraline_2000,
	title = {Sertraline induced hypomania},
	volume = {42},
	number = {1},
	journal = {Indian Journal of Psychiatry},
	author = {R. Kumar and C. Dubey and V. Sinha},
	year = {2000},
	pages = {104}
},

@article{mcdaniel_possible_1990,
	title = {Possible induction of mania by buspirone},
	volume = {147},
	number = {1},
	journal = {American Journal of Psychiatry},
	author = {J. S. {McDaniel} and P. T. Ninan and J. V. Magnuson},
	year = {1990},
	pages = {125}
},

@article{venkataraman_mania_1992-1,
	title = {Mania associated with fluoxetine treatment in adolescents},
	volume = {31},
	journal = {J Am Acad Child Adolesc Psychiatry},
	author = {S. Venkataraman and M. W. Naylor and C. A. King},
	year = {1992},
	pages = {276--281}
},

@article{partin_tachyarrhythmia_2004,
	title = {Tachyarrhythmia and Hypomania With Horny Goat Weed},
	volume = {45},
	url = {http://psy.psychiatryonline.org},
	doi = {10.1176/appi.psy.45.6.536},
	number = {6},
	journal = {Psychosomatics},
	author = {Jessica F. Partin and Yaacov R. Pushkin},
	month = dec,
	year = {2004},
	pages = {536--537}
},

@article{perlis_serotonin_2003-1,
	title = {Serotonin transporter polymorphisms and adverse effects with fluoxetine treatment},
	volume = {54},
	number = {9},
	journal = {Biological Psychiatry},
	author = {R. H. Perlis and D. Mischoulon and J. W. Smoller and Y. J. Y. Wan and S. {Lamon-Fava} and K. M. Lin and J. F. Rosenbaum and M. Fava},
	year = {2003},
	pages = {879--883}
},

@article{baldassano_ziprasidone-associated_2003,
	title = {Ziprasidone-associated mania: a case series and review of the mechanism},
	volume = {5},
	number = {1},
	journal = {Bipolar Disorders},
	author = {C. F. Baldassano and C. Ballas and S. M. Datto and D. Kim and L. Littman and J. {O'Reardon} and M. A. Rynn},
	year = {2003},
	pages = {72--75}
},

@article{peet_induction_1994-1,
	title = {Induction of mania with selective serotonin re-uptake inhibitors and tricyclic antidepressants},
	volume = {164},
	number = {4},
	journal = {The British Journal of Psychiatry},
	author = {M. Peet},
	year = {1994},
	pages = {549--550}
},

@article{amsterdam_efficacy_1998,
	title = {Efficacy and Safety of Fluoxetine in Treating Bipolar {II} Major Depressive Episode.},
	volume = {18},
	number = {6},
	journal = {Journal of clinical psychopharmacology},
	author = {J. D. Amsterdam and F. {Garcia-Espana} and J. Fawcett and F. M. Quitkin and F. W. Reimherr and J. F. Rosenbaum and E. Schweizer and C. Beasley},
	year = {1998},
	pages = {435}
},

@article{cohen_implications_1978,
	title = {Implications of monoamine oxidase inhibitor-induced hypomania.},
	volume = {35},
	number = {11},
	journal = {Archives of general psychiatry},
	author = {R. Cohen and D. Pickar},
	year = {1978},
	pages = {1393}
},

@article{simon_hypomania-like_1999,
	title = {Hypomania-like syndrome induced by olanzapine.},
	volume = {14},
	number = {6},
	journal = {International clinical psychopharmacology},
	author = {A. E. Simon and J. M. Aubry and L. Malky and G. Bertschy},
	year = {1999},
	pages = {377}
},

@article{gualtieri_paradoxical_1991,
	title = {Paradoxical Effects of Fluoxetine.},
	volume = {11},
	number = {6},
	journal = {Journal of Clinical Psychopharmacology},
	author = {{CT} {GUALTIERI}},
	year = {1991},
	pages = {393}
},

@article{michael_acute_2003,
	title = {Acute mania is accompanied by elevated glutamate/glutamine levels within the left dorsolateral prefrontal cortex},
	volume = {168},
	url = {http://dx.doi.org/10.1007/s00213-003-1440-z},
	doi = {10.1007/s00213-003-1440-z},
	abstract = {Abstract
Rationale  The dorsolateral prefrontal cortex {(DLPFC)} participates in the pathophysiology of mania. In particular, left-sided structural
and metabolic abnormalities have been described.



Objectives  Clinical symptoms may be due to hyperactivity of cortical glutamatergic neurons, resulting in increased excitatory neurotransmitter
flux and thus enhanced Glx levels.



Methods  Glutamate/glutamine {(Glx)} levels were assessed by proton magnetic resonance spectroscopy {(1H-MRS)} in eight acute manic patients compared with age- and gender-matched controls.



Results  Manic patients had significantly elevated Glx levels (t-test; t=–3.1, P=0.008) within the left {DLPFC.}



Conclusions  Our results indicate that the prefrontal cortical glutamatergic system is involved in the pathophysiology of acute mania.
This may have implications for the treatment of mania.


},
	number = {3},
	journal = {Psychopharmacology},
	author = {Nikolaus Michael and Andreas Erfurth and Patricia Ohrmann and Michael Gössling and Volker Arolt and Walter Heindel and Bettina Pfleiderer},
	month = jul,
	year = {2003},
	pages = {344--346}
},

@article{hougland_positron_2008,
	title = {Positron emission tomography with {fluorodeoxyglucose-F18} in an animal model of mania},
	volume = {164},
	issn = {0925-4927},
	url = {http://www.sciencedirect.com/science/article/B6TBW-4TPWVWY-4/2/eb7b0e96fdcb3fbe47219947bad13868},
	doi = {10.1016/j.pscychresns.2008.01.004},
	abstract = {
Intracerebroventricular {(ICV)} administration of ouabain to young adult rats has been suggested to model human bipolar mania. In the human condition, mania and bipolar depression are both associated with reductions in frontal cerebral metabolism. We utilized {[18F]-fluorodeoxyglucose} {[18FDG]} positron emission tomography {(PET)} to visualize glucose uptake in animals receiving {ICV} ouabain. Animals received 5 µl of 10- 3 M ouabain {ICV,} were anesthetized with isoflurane inhalation, and administered intraperitoneally with 0.5 {mCi} of {18FDG.} {PET} data were collected over 20 min 1 hour later. Additionally, the effect of lithium was examined in animals receiving lithium in their diet for 1 week before the {ICV} ouabain injection. Data were analyzed with {IDL} Virtual Machine software. Brain glucose utilization as measured by {18FDG} uptake was significantly reduced in animals receiving {ICV} ouabain compared with those receiving equal volumes of artificial cerebrospinal fluid. Pretreatment with lithium normalized {18FDG} uptake. These results mirror human studies.},
	number = {2},
	journal = {Psychiatry Research: Neuroimaging},
	author = {Matthew Tyler Hougland and Yonglin Gao and Laura Herman and Chin K. Ng and Zhenmin Lei and Rif S. {El-Mallakh}},
	month = nov,
	year = {2008},
	keywords = {{[18F]-Fluorodeoxyglucose} {[18FDG],Animal} {model,Bipolar} {disorder,Cerebral} {metabolism,Glucose} {uptake,microPET,Ouabain,Rat}},
	pages = {166--171}
},

@article{harsch_induction_1985,
	title = {Induction of mania by L-dopa in a nonbipolar patient.},
	volume = {5},
	number = {6},
	journal = {Journal of clinical psychopharmacology},
	author = {H. H. Harsch and M. Miller and L. D. Young},
	year = {1985},
	pages = {338}
},

@article{colombo_rate_1999,
	title = {Rate of switch from depression into mania after therapeutic sleep deprivation in bipolar depression},
	volume = {86},
	number = {3},
	journal = {Psychiatry Research},
	author = {C. Colombo and F. Benedetti and B. Barbini and E. Campori and E. Smeraldi},
	year = {1999},
	pages = {267--270}
},

@article{joffe_induction_2002,
	title = {Induction of mania and cycle acceleration in bipolar disorder: effect of different classes of antidepressant},
	volume = {105},
	number = {6},
	journal = {Acta Psychiatrica Scandinavica},
	author = {R. T. Joffe and G. M. {MacQueen} and M. Marriott and J. Robb and H. Begin and L. T. Young},
	year = {2002},
	pages = {427--430}
},

@article{sakkas_induction_2003-1,
	title = {Induction of mania by {rTMS:} report of two cases},
	volume = {18},
	number = {4},
	journal = {European Psychiatry},
	author = {P. Sakkas and P. Mihalopoulou and P. Mourtzouhou and C. Psarros and V. Masdrakis and A. Politis and G. N. Christodoulou},
	year = {2003},
	pages = {196--198}
},

@article{damore_medication-induced_1998-1,
	title = {Medication-induced hypomania in Asperger's disorder},
	volume = {37},
	number = {3},
	journal = {Journal of the American Academy of Child \& Adolescent Psychiatry},
	author = {J. Damore and J. Stine and L. Brody},
	year = {1998},
	pages = {248--249}
},

@article{dubovsky_calcium_1993,
	title = {Calcium antagonists in manic-depressive illness},
	volume = {27},
	journal = {Neuropsychobiology},
	author = {S. L. Dubovsky},
	year = {1993},
	pages = {184--192}
},

@article{pfohl_mathematical_1981,
	title = {The mathematical case against unipolar mania.},
	volume = {16},
	number = {4},
	journal = {Journal of psychiatric research},
	author = {B. Pfohl and N. Vasquez and H. Nasrallah},
	year = {1981},
	pages = {259}
},

@article{martinez-aran_cognitive_2004,
	title = {Cognitive Function Across Manic or Hypomanic, Depressed, and Euthymic States in Bipolar Disorder},
	volume = {161},
	url = {http://ajp.psychiatryonline.org/cgi/content/abstract/161/2/262},
	doi = {10.1176/appi.ajp.161.2.262},
	abstract = {{OBJECTIVE:} The study aims were to address neuropsychological functioning across different states of bipolar illness and to determine relationships among clinical features, neuropsychological performance, and psychosocial functioning. {METHOD:} Several domains of cognitive function were examined in 30 depressed bipolar patients {(DSM-IV} criteria for major depression, Hamilton Depression Rating Scale score {[IMG]="} {BORDER="0"{\textgreater}17),} 34 manic or hypomanic bipolar patients {(DSM-IV} criteria for manic or hypomanic episode, Young Mania Rating Scale score {[IMG]="} {BORDER="0"{\textgreater}12),} and 44 euthymic bipolar patients (6 months of remission, Hamilton depression scale score [{\textless}=]8, and Young Mania Rating Scale score [{\textless}=]6). The comparison group consisted of 30 healthy subjects without history of neurological or psychiatric disorders. A neuropsychological battery assessed executive function, attention, and verbal and visual memory. {RESULTS:} The three groups showed cognitive dysfunction in verbal memory and frontal executive tasks in relation to the comparison group. Low neuropsychological performance was associated with poor functional outcome. Impairment of verbal memory was related to the duration of illness and the numbers of previous manic episodes, hospitalizations, and suicide attempts. {CONCLUSIONS:} A poorer performance was observed in all bipolar groups regarding executive function and verbal memory in relation to the healthy comparison subjects. These cognitive difficulties, especially related to verbal memory, may help explain the impairment regarding daily functioning, even during remission. Further studies should focus on testing, whether optimizing prophylactic pharmacological treatment and psychoeducation might reduce cognitive impairment, and whether bipolar patients would benefit from neuropsychological rehabilitation in order to reduce the impact of cognitive impairment in their overall functioning.
},
	number = {2},
	journal = {Am J Psychiatry},
	author = {Anabel {Martinez-Aran} and Eduard Vieta and Maria Reinares and Francesc Colom and Carla Torrent and Jose {Sanchez-Moreno} and Antonio Benabarre and Jose Manuel Goikolea and Merce Comes and Manel Salamero},
	month = feb,
	year = {2004},
	pages = {262--270}
},

@article{benazzi_atypical_2001,
	title = {Atypical depression with hypomanic symptoms},
	volume = {65},
	number = {2},
	journal = {Journal of Affective Disorders},
	author = {F. Benazzi},
	year = {2001},
	pages = {179--183}
},

@article{murphy_regular_1971,
	title = {Regular Induction of Hypomania by {L-Dopa} in {[ldquo]Bipolar[rdquo]} Manic-depressive Patients},
	volume = {229},
	url = {http://dx.doi.org/10.1038/229135a0},
	doi = {10.1038/229135a0},
	number = {5280},
	journal = {Nature},
	author = {{DENNIS} L. {MURPHY} and H. {KEITH} H. {BRODIE} and {FREDERICK} K. {GOODWIN} and {WILLIAM} E. {BUNNEY}},
	year = {1971},
	pages = {135--136}
},

@article{chouinard_case_1986,
	title = {A case of mania induced by high-dose fluoxetine treatment.},
	volume = {143},
	number = {5},
	journal = {Am J Psychiatry},
	author = {G. Chouinard and W. Steiner},
	year = {1986},
	pages = {686}
},

@article{cipriani_haloperidol_????,
	title = {Haloperidol alone or in combination for acute mania {(Review)}},
	author = {A. Cipriani and J. M. Rendell and J. R. Geddes}
},

@article{strmberg_l-dopa_1971,
	title = {{l-DOPA} induced effects on motor activity in mice after inhibition of dopamine-β-hydroxylase},
	volume = {19},
	url = {http://dx.doi.org/10.1007/BF00403702},
	doi = {{10.1007/BF00403702}},
	abstract = {The modification by {FLA-63} bis-(4-methyl-1-homopiperazinyl thiocarbonyl)-disulfide {(Florvall} and Corrodi, 1970), an inhibitor of dopamine {(DA)-ß-hydroxylase,} of the effect of different doses of {l-DOPA} on motor activity and monoamine levels in the caudate nucleus and the rest of the brain, was studied. Throughout the experiment extracerebral decarboxylase was inhibited by means of Mk-485. {FLA-63} did not alter the depressant effect on motility of a medium dose of {l-DOPA,} although the characteristic hyperexcitability was abolished. However, the strong motor stimulant effect of a high dose of {l-DOPA} was partly antagonized by {FLA-63.} {l-DOPA} caused an accumulation of {DA} both in the caudate nucleus and in the rest of the brain, a small but significant decrease in central noradrenaline {(NA)} and a decrease in 5-hydroxytryptamine in both brain parts. This pattern was mainly unaffected by {FLA-63,} which by itself caused a decrease in {NA.} It was concluded, that both {NA} and {DA} are propably of importance for the motor stimulant effect of {l-DOPA} in the mouse.},
	number = {1},
	journal = {Psychopharmacology},
	author = {U. Strömberg and T. H. Svensson},
	month = mar,
	year = {1971},
	pages = {53--60}
},

@article{dummit_fluoxetine_1996,
	title = {Fluoxetine treatment of children with selective mutism: an open trial},
	volume = {35},
	number = {5},
	journal = {Journal of the American Academy of Child and Adolescent Psychiatry},
	author = {E. S. Dummit and R. G. Klein and N. K. Tancer and B. Asche and J. Martin},
	year = {1996},
	pages = {615--621}
},

@article{pae_fluoxetine-induced_2004,
	title = {Fluoxetine-induced mania in an Asian patient.},
	volume = {58},
	number = {4},
	journal = {Psychiatry \& Clinical Neurosciences},
	author = {{CHIUN} {PAE} and {CUK} {LEE} and {SOOJ} {LEE} and C. {LEE} and {INHO} {PAIK}},
	year = {2004},
	pages = {448}
},

@article{aghanwa_recurrent_2001,
	title = {Recurrent unipolar mania in a psychiatric hospital setting in the Fiji Islands},
	volume = {34},
	journal = {Psychopathology},
	author = {H. S. Aghanwa},
	year = {2001},
	pages = {312--317}
},

@article{lange-asschenfeldt_serotonin_2002,
	title = {Serotonin Syndrome as a Result of Fluoxetine in a Patient With Tramadol Abuse: Plasma Level-correlated Symptomatology.},
	volume = {22},
	number = {4},
	journal = {Journal of clinical psychopharmacology},
	author = {C. {Lange-Asschenfeldt} and H. Weigmann and C. Hiemke and K. Mann},
	year = {2002},
	pages = {440}
},

@article{delbello_magnetic_2004,
	title = {Magnetic resonance imaging analysis of amygdala and other subcortical brain regions in adolescents with bipolar disorder},
	volume = {6},
	number = {1},
	journal = {Bipolar Disorders},
	author = {M. P. {DelBello} and M. E. Zimmerman and N. P. Mills and G. E. Getz and S. M. Strakowski},
	year = {2004},
	pages = {43--52}
},

@article{abler_abnormal_2007,
	title = {Abnormal Reward System Activation in Mania},
	volume = {33},
	issn = {{0893-133X}},
	url = {http://dx.doi.org/10.1038/sj.npp.1301620},
	number = {9},
	journal = {Neuropsychopharmacology},
	author = {Birgit Abler and Ian Greenhouse and Dost Ongur and Henrik Walter and Stephan Heckers},
	month = nov,
	year = {2007},
	pages = {2217--2227}
},

@article{sotsky_tolmetin_1984,
	title = {Tolmetin induction of mania},
	volume = {25},
	number = {8},
	journal = {Psychosomatics},
	author = {{SM} {SOTSKY} and {JW} {TOSSELL}},
	year = {1984},
	pages = {626--628}
},

@article{azab_lipid_2006,
	title = {Lipid connection to bipolar disorder},
	volume = {1},
	number = {4},
	journal = {Future Neurol.},
	author = {A. N. Azab and M. L. Greenberg},
	year = {2006},
	pages = {505--513}
},

@article{przedborski_induction_1992,
	title = {Induction of mania by apomorphine in a depressed parkinsonian patient},
	volume = {7},
	number = {3},
	journal = {Movement Disorders},
	author = {S. Przedborski and A. Liard and J. Hildebrand},
	year = {1992}
},

@article{aubry_possible_2000,
	title = {Possible induction of mania and hypomania by olanzapine or risperidone: a critical review of reported cases.},
	volume = {61},
	number = {9},
	journal = {The Journal of clinical psychiatry},
	author = {J. M. Aubry and A. E. Simon and G. Bertschy},
	year = {2000},
	pages = {649}
},

@article{howland_induction_1996,
	title = {Induction of Mania With Serotonin Reuptake Inhibitors.},
	volume = {16},
	number = {6},
	journal = {Journal of clinical psychopharmacology},
	author = {R. H. Howland},
	year = {1996},
	pages = {425}
},

@book{su_are_2000,
	title = {Are ?3 Fatty Acids Beneficial in Depression but Not Mania?},
	volume = {57},
	publisher = {Am Med Assoc},
	author = {K. P. Su and W. W. Shen and S. Y. Huang and A. L. Stoll and K. E. Damico and L. B. Marangell and W. E. Severus},
	year = {2000},
	pages = {716--717}
},

@book{horrobin_omega-3_2003,
	title = {Omega-3 fatty acid for schizophrenia},
	volume = {160},
	publisher = {Am Psychiatric Assoc},
	author = {{DF} {HORROBIN}},
	year = {2003},
	pages = {188--189}
},

@article{lavin_adverse_1993-1,
	title = {Adverse Reaction to {High-Dose} Fluoxetine.},
	volume = {13},
	number = {6},
	journal = {Journal of clinical psychopharmacology},
	author = {M. R. Lavin and A. Mendelowitz and S. H. Block},
	year = {1993},
	pages = {452}
},

@article{duggal_bipolar_2003,
	title = {Bipolar Disorder With Asperger's Disorder},
	volume = {160},
	url = {http://ajp.psychiatryonline.org},
	doi = {10.1176/appi.ajp.160.1.184-a},
	number = {1},
	journal = {Am J Psychiatry},
	author = {{HARPREET} S. {DUGGAL}},
	year = {2003},
	pages = {184--a-185}
},

@book{chang_anomalous_2004,
	title = {Anomalous {Prefrontal-Subcortical} Activation in Familial Pediatric Bipolar Disorder A Functional Magnetic Resonance Imaging Investigation},
	volume = {61},
	publisher = {Am Med Assoc},
	author = {K. Chang and N. E. Adleman and K. Dienes and D. I. Simeonova and V. Menon and A. Reiss},
	year = {2004},
	pages = {781--792}
},

@article{mandalos_dose-related_1990,
	title = {{Dose-Related} Paranoid Reaction Associated with Fluoxetine.},
	volume = {178},
	number = {1},
	journal = {The Journal of Nervous and Mental Disease},
	author = {G. E. Mandalos and B. L. Szarek},
	year = {1990},
	pages = {57}
},

@article{piredda_hypomania_1992,
	title = {Hypomania induced by fluoxetine?},
	volume = {32},
	number = {1},
	journal = {Biol Psychiatry},
	author = {S. G. Piredda and S. L. Rubinstein},
	year = {1992},
	pages = {107}
},

@article{shulman_unipolar_1994,
	title = {Unipolar mania reconsidered: evidence from an elderly cohort},
	volume = {164},
	number = {4},
	journal = {The British Journal of Psychiatry},
	author = {K. I. Shulman},
	year = {1994},
	pages = {547--549}
},

@article{murphy_catecholamines_1972,
	title = {Catecholamines and memory: Enhanced verbal learning during {l-DOPA} administration},
	volume = {27},
	url = {http://dx.doi.org/10.1007/BF00429385},
	doi = {{10.1007/BF00429385}},
	abstract = {Verbal learning was measured during the administration of {l-DOPA} in large oral doses to depressed patients. Longer-term memory on two different tasks improved during treatment, while short-term memory (immediate recall) was unaffected. In contrast, the catecholamine synthesis inhibitor a-methyl-p-tyrosine did not alter either memory process.},
	number = {4},
	journal = {Psychopharmacology},
	author = {Dennis L. Murphy and George M. Henry and Herbert Weingartner},
	month = dec,
	year = {1972},
	pages = {319--326}
},

@article{beech_sertraline-induced_2000,
	title = {Sertraline-induced hypomania presenting as hyperactivity disorder.},
	volume = {36},
	number = {3},
	journal = {Journal of Paediatrics \& Child Health},
	author = {M. J. Beech},
	year = {2000},
	pages = {290}
},

@article{damore_medication-induced_1998-2,
	title = {Medication-induced hypomania in Asperger's disorder.},
	volume = {37},
	number = {3},
	journal = {Journal of the American Academy of Child and Adolescent Psychiatry},
	author = {J. Damore and J. Stine and L. Brody},
	year = {1998},
	pages = {248}
},

@article{sonawalla_fluoxetine_2002,
	title = {Fluoxetine treatment of depressed patients with comorbid anxiety disorders},
	volume = {16},
	number = {3},
	journal = {Journal of Psychopharmacology},
	author = {S. B. Sonawalla and A. Farabaugh and M. W. Johnson and M. Morray and M. L. Delgado and M. G. Pingol and J. F. Rosenbaum and M. Fava},
	year = {2002},
	pages = {215}
},

@book{mundo_role_2001,
	title = {The Role of Serotonin Transporter Protein Gene in {Antidepressant-Induced} Mania in Bipolar Disorder Preliminary Findings},
	volume = {58},
	publisher = {Am Med Assoc},
	author = {E. Mundo and M. Walker and T. Cate and F. Macciardi and J. L. Kennedy},
	year = {2001},
	pages = {539--544}
},

@article{dubovsky_phenelzine-induced_1985,
	title = {Phenelzine-induced hypomania: effect of verapamil.},
	volume = {20},
	number = {9},
	journal = {Biological psychiatry},
	author = {S. L. Dubovsky and R. D. Franks and D. Schrier},
	year = {1985},
	pages = {1009}
},

@article{volpe_seasonality_2006,
	title = {Seasonality of admissions for mania in a psychiatric hospital of Belo Horizonte, Brazil},
	volume = {94},
	number = {1-3},
	journal = {Journal of Affective Disorders},
	author = {F. M. Volpe and J. A. Del Porto},
	year = {2006},
	pages = {243--248}
},

@article{frye_reduced_2007,
	title = {Reduced concentrations of N-acetylaspartate {(NAA)} and the {NAA–creatine} ratio in the basal ganglia in bipolar disorder: A study using {3-Tesla} proton magnetic resonance spectroscopy},
	volume = {154},
	number = {3},
	journal = {Psychiatry Research: Neuroimaging},
	author = {M. A. Frye and M. A. Thomas and K. Yue and N. Binesh and P. Davanzo and J. Ventura and J. {O'Neill} and B. Guze and J. G. Curran and J. Mintz},
	year = {2007},
	pages = {259--265}
},

@article{mendhekar_aripiprazole-induced_2004,
	title = {Aripiprazole-induced rabbit syndrome.},
	volume = {38},
	number = {7},
	journal = {Australian \& New Zealand Journal of Psychiatry},
	author = {D. N. Mendhekar},
	year = {2004},
	pages = {561}
},

@article{obrien_mental_1971,
	title = {Mental Effects of {High-Dosage} Levodopa},
	volume = {24},
	url = {http://archpsyc.ama-assn.org/cgi/content/abstract/24/1/61},
	doi = {10.1001/archpsyc.24.1.61},
	abstract = {Of 200 patients receiving levodopa {(L-DOPA)} therapy the first 20 Patients received 4 to 6.5 gm of {LDOPA} per day. Seven of 12 Parkinsonian patients who were also depressed showed a remission of depression during {L-DOPA} treatment, however this change generally correlated with the degree of motor improvement. In two patients the mood elevation was clearly out of proportion to the motor improvement, and one of these developed a "hypomanic-like" state. A third Patient developed agitated behavior after one year of {L-DOPA} therapy. Six of nine males reported spontaneous penile erections and one resumed successful intercourse after three years of impotency. Of four patients with organic dementia, three showed no detectable improvement but one raised his Wechsler Memory Quotient from 59 to 86.
},
	number = {1},
	journal = {Arch Gen Psychiatry},
	author = {Charles P. {O'Brien} and Joseph N. {DiGiacomo} and Stanley. Fahn and Gabriel A. Schwarz},
	year = {1971},
	pages = {61--64}
},

@article{howland_induction_1996-1,
	title = {Induction of Mania With Serotonin Reuptake Inhibitors.},
	volume = {16},
	number = {6},
	journal = {Journal of clinical psychopharmacology},
	author = {R. H. Howland},
	year = {1996},
	pages = {425}
},

@article{vairo_high_2004,
	title = {{HIGH} {DOSE} {FLUOXETINE-INDUCED} {MANIA:} {BIBLIOGRAPHIC} {REVIEW} {AND}},
	journal = {Trends in Bipolar Disorder Research},
	author = {M. C. Vairo and M. Ruiz and D. Matusevich and C. Finkelsztein},
	year = {2004},
	pages = {153}
},

@article{horiguchi_display_2001,
	title = {A Display of Hypomania in a Depressed Male in Response to Fluvoxamine},
	volume = {2},
	journal = {Antidepressant Drug Discovery in the Postgenomic Era},
	author = {T. Horiguchi and S. Sai},
	year = {2001},
	pages = {201--204}
},

@article{horiguchi_display_2001-1,
	title = {A Display of Hypomania in a Depressed Male in Response to Fluvoxamine},
	volume = {2},
	journal = {Antidepressant Drug Discovery in the Postgenomic Era},
	author = {T. Horiguchi and S. Sai},
	year = {2001},
	pages = {201--204}
}