@article{alexander_parallel_1986,
author = {Alexander, G E and DeLong, M R and Strick, P L},
doi = {10.1146/annurev.ne.09.030186.002041},
issn = {0147-006X},
journal = {Annual Review of Neuroscience},
keywords = {{Basal} {Ganglia,Behavior,Brain} {Mapping,Cats,Cerebral} {Cortex,Corpus} {Striatum,Haplorhini,Movement,Nervous} System {Diseases,Neural} {Pathways,Septum} {Pellucidum,Substantia} {Nigra,Thalamus,Visual} Pathways,{Animals,} {Animal}},
note = {{PMID:} 3085570
},
pages = {357--381},
title = {Parallel organization of functionally segregated circuits linking basal ganglia and cortex},
url = {http://www.ncbi.nlm.nih.gov/pubmed/3085570},
volume = {9},
year = {1986}
}
@article{alm_stuttering_2006,
author = {Alm, P A},
journal = {Brain and Language},
pages = {317--321},
title = {Stuttering and sensory gating: A study of acoustic startle prepulse inhibition},
volume = {97},
year = {2006}
}
@article{barnier_retrieval-induced_2004,
author = {Barnier, A J and Hung, L and Conway, M A},
journal = {Emotional Memory Failures},
pages = {457},
title = {Retrieval-induced forgetting of emotional and unemotional autobiographical memories},
year = {2004}
}
@article{buml_markov_1996,
author = {Bauml, K H},
journal = {Acta Psychologica},
pages = {231--250},
title = {A Markov model for measuring storage loss and retrieval failure in retroactive inhibition},
volume = {92},
year = {1996}
}
@article{buml_oscillatory_2008,
author = {Bauml, K H and Hanslmayr, S and Pastötter, B and Klimesch, W},
journal = {Neuroimage},
pages = {596--604},
title = {Oscillatory correlates of intentional updating in episodic memory},
volume = {41},
year = {2008}
}
@article{benjamin_effects_2006,
author = {Benjamin, A S},
journal = {{PSYCHONOMIC} {BULLETIN} {AND} {REVIEW}},
pages = {831},
title = {The effects of list-method directed forgetting on recognition memory},
volume = {13},
year = {2006}
}
@article{bjork_intentional_2003,
author = {Bjork, E L and Bjork, R A},
journal = {Learning, Memory},
pages = {524--531},
title = {Intentional forgetting can increase, not decrease, residual influences of to-be-forgotten information},
volume = {29},
year = {2003}
}
@article{brene_expression_1994,
author = {Brene, S and Lindefors, N and Ehrlich, M and Taubes, T and Horiuchi, A and Kopp, J and Hall, H and Sedvall, G and Greengard, P and PERSSON, H},
journal = {J. Neurosci.},
pages = {985--998},
title = {Expression of {mRNAs} encoding {ARPP-16/19,} {ARPP-21,} and {DARPP-32} in human brain tissue},
url = {http://www.jneurosci.org/cgi/content/abstract/14/3/985},
volume = {14},
year = {1994}
}
@article{centonze_dopaminergic_2001,
abstract = {Cortical glutamatergic and nigral dopaminergic afferents impinge on projection spiny neurons of the striatum, providing the most significant inputs to this structure. Isolated activation of glutamate or dopamine {(DA)} receptors produces short-term effects on striatal neurons, whereas the combined stimulation of both glutamate and {DA} receptors is able to induce long-lasting modifications of synaptic excitability. Repetitive stimulation of corticostriatal fibres causes a massive release of both glutamate and {DA} in the striatum and, depending on the glutamate receptor subtype preferentially activated, produces either long-term depression {(LTD)} or long-term potentiation {(LTP)} of excitatory synaptic transmission. D1-like and D2-like {DA} receptors interact synergistically to allow {LTD} formation, while they operate in opposition during the induction phase of {LTP.} Corticostriatal synaptic plasticity is severely impaired after chronic {DA} denervation and requires the stimulation of {DARPP-32,} a small protein expressed in dopaminoceptive spiny neurons which acts as a potent inhibitor of protein phosphatase-1. In addition, the formation of {LTD} and {LTP} requires the activation of {PKG} and {PKA,} respectively, in striatal projection neurons. These kinases appear to be stimulated by the activation of D1-like receptors in distinct neuronal populations.},
author = {Centonze, D and Picconi, B and Gubellini, P and Bernardi, G and Calabresi, P},
issn = {0953-816X},
journal = {The European Journal of Neuroscience},
keywords = {{Corpus} {Striatum,Dopamine,Dopamine} and {cAMP-Regulated} Phosphoprotein {32,Long-Term} {Potentiation,Nerve} Tissue {Proteins,Neuronal} {Plasticity,Phosphoproteins,Receptors,Synapses,Synaptic} Transmission,{Animals,} Dopamine {D1,} Dopamine {D2}},
note = {{PMID:} 11285003
},
pages = {1071--1077},
title = {Dopaminergic control of synaptic plasticity in the dorsal striatum},
url = {http://www.ncbi.nlm.nih.gov/pubmed/11285003},
volume = {13},
year = {2001}
}
@article{cooke_protein_2002,
author = {Cooke, S F},
journal = {Trends in Neurosciences},
pages = {606--607},
title = {Protein phosphatases: a means of forgetting},
volume = {25},
year = {2002}
}
@article{cooke_genetic_2003,
author = {Cooke, S F and Bliss, T V P},
journal = {Cellular and Molecular Life Sciences {(CMLS)}},
pages = {1--5},
title = {The genetic enhancement of memory},
volume = {60},
year = {2003}
}
@article{dunnett_frontal-striatal_2005,
abstract = {The prefrontal cortex is considered to provide executive control of response selection and planning in diverse cognitive tasks, translated into action via descending subcortical projections (or 'loops') through the basal ganglia. We have used a disconnection strategy to demonstrate first that bilateral fronto-striatal disconnection disrupts rats' abilities to perform delayed alternation, the classic test of prefrontal function in rats and monkeys, and second that crossed unilateral cortical and striatal lesions on opposite sides similarly disrupt rats' abilities to perform the same cognitive task. We found that effective disconnection requires interruption of interhemispheric transfer, achieved by transection of the anterior corpus callosum. This produces a moderate deficit in its own right, which is not exacerbated by additional prefrontal and striatal lesions in one hemisphere. Conversely, the animals are significantly more impaired after crossed prefrontal and striatal lesions of similar total magnitude. The results demonstrate than an intact cortico-striatal pathway is necessary to sustain performance on a classical prefrontal task, and provide a model within which to assess circuit reconstruction with novel cell therapies for brain repair.},
author = {Dunnett, S B and Meldrum, A and Muir, J L},
doi = {10.1016/j.neuroscience.2005.07.033},
issn = {0306-4522},
journal = {Neuroscience},
keywords = {{Conditioning,Corpus} {Striatum,Functional} {Laterality,Male,Neural} {Pathways,Prefrontal} {Cortex,Rats},{Animals,} {Operant}},
note = {{PMID:} 16165288
},
pages = {1055--1065},
title = {Frontal-striatal disconnection disrupts cognitive performance of the frontal-type in the rat},
url = {http://www.ncbi.nlm.nih.gov/pubmed/16165288},
volume = {135},
year = {2005}
}
@article{fienberg_darpp-32:_1998,
author = {Fienberg, A A and Hiroi, N and Mermelstein, P G and Song, W.-J. and Snyder, G L and Nishi, A and Cheramy, A and O'callaghan, J P and Miller, D B and Cole, D G and Corbett, R and Haile, C N and Cooper, D C and Onn, S P and Grace, A A and Ouimet, C C and White, F J and Hyman, S E and Surmeier, D J and Girault, J.-A. and Nestler, E J and Greengard, P},
doi = {10.1126/science.281.5378.838},
journal = {Science},
pages = {838--842},
title = {{DARPP-32:} Regulator of the Efficacy of Dopaminergic Neurotransmission},
url = {http://www.sciencemag.org/cgi/content/abstract/281/5378/838},
volume = {281},
year = {1998}
}
@article{fleck_processing_2005,
author = {Fleck, D E and Shear, P K and Strakowski, S M},
journal = {Journal of the International Neuropsychological Society},
pages = {871--880},
title = {Processing efficiency and directed forgetting in bipolar disorder},
volume = {11},
year = {2005}
}
@article{foster_individual_????,
author = {Foster, N L and Sahakyan, L},
title = {{INDIVIDUAL} {DIFFERENCES} {IN} {FORGETTING} {STRATEGIES}}
}
@article{foster_calcium_2002,
author = {Foster, T C and Kumar, A},
journal = {The Neuroscientist},
pages = {297},
title = {Calcium dysregulation in the aging brain},
volume = {8},
year = {2002}
}
@article{golding_recall_2005,
author = {Golding, J M and Gottlob, L R},
journal = {Memory and Cognition},
pages = {588},
title = {Recall order determines the magnitude of directed forgetting in the within-participants list method},
volume = {33},
year = {2005}
}
@article{gottlob_directed_2006,
author = {Gottlob, L R and Golding, J M and Hauselt, W J},
journal = {The Journal of general psychology},
pages = {67--80},
title = {Directed forgetting of a single item},
volume = {133},
year = {2006}
}
@article{gray_neural_2003,
abstract = {We used an individual-differences approach to test whether general fluid intelligence {(gF)} is mediated by brain regions that support attentional (executive) control, including subregions of the prefrontal cortex. Forty-eight participants first completed a standard measure of {gF} {(Raven's} Advanced Progressive Matrices). They then performed verbal and nonverbal versions of a challenging working-memory task (three-back) while their brain activity was measured using functional magnetic resonance imaging {(fMRI).} Trials within the three-back task varied greatly in the demand for attentional control because of differences in trial-to-trial interference. On high-interference trials specifically, participants with higher {gF} were more accurate and had greater event-related neural activity in several brain regions. Multiple regression analyses indicated that lateral prefrontal and parietal regions may mediate the relation between ability {(gF)} and performance (accuracy despite interference), providing constraints on the neural mechanisms that support {gF.}},
author = {Gray, Jeremy R and Chabris, Christopher F and Braver, Todd S},
doi = {10.1038/nn1014},
issn = {1097-6256},
journal = {Nature Neuroscience},
keywords = {{Adult,Attention,Brain,Brain} {Mapping,Choice} {Behavior,Cognition,Conflict} {(Psychology),Female,Humans,Intelligence,Intelligence} {Tests,Magnetic} Resonance {Imaging,Male,Memory,Multivariate} {Analysis,Parietal} {Lobe,Predictive} Value of {Tests,Prefrontal} {Cortex,Problem} {Solving,Reference} {Values,Regression} {Analysis,Verbal} Behavior,{Adolescent,} {Short-Term}},
note = {{PMID:} 12592404
},
pages = {316--322},
title = {Neural mechanisms of general fluid intelligence},
url = {http://www.ncbi.nlm.nih.gov/pubmed/12592404},
volume = {6},
year = {2003}
}
@article{graybiel_basal_2005,
abstract = {The field of basal ganglia research is exploding on every level - from discoveries at the molecular level to those based on human brain imaging. A remarkable series of new findings support the view that the basal ganglia are essential for some forms of learning-related plasticity. Other new findings are challenging some of the basic tenets of the field as it now stands. Combined with the new evidence on learning-related functions of the basal ganglia, these studies suggest that the basal ganglia are parts of a brain-wide set of adaptive neural systems promoting optimal motor and cognitive control.},
author = {Graybiel, Ann M},
doi = {10.1016/j.conb.2005.10.006},
issn = {0959-4388},
journal = {Current Opinion in Neurobiology},
keywords = {{Basal} {Ganglia,Cerebellum,Cerebral} {Cortex,Efferent} {Pathways,Humans,Learning,Neuronal} {Plasticity,Reinforcement} {(Psychology)},{Animals}},
note = {{PMID:} 16271465
},
pages = {638--644},
title = {The basal ganglia: learning new tricks and loving it},
url = {http://www.ncbi.nlm.nih.gov/pubmed/16271465},
volume = {15},
year = {2005}
}
@article{haber_primate_2003,
abstract = {The basal ganglia and frontal cortex operate together to execute goal directed behaviors. This requires not only the execution of motor plans, but also the behaviors that lead to this execution, including emotions and motivation that drive behaviors, cognition that organizes and plans the general strategy, motor planning, and finally, the execution of that plan. The components of the frontal cortex that mediate these behaviors, are reflected in the organization, physiology, and connections between areas of frontal cortex and in their projections through basal ganglia circuits. This comprises a series of parallel pathways. However, this model does not address how information flows between circuits thereby developing new learned behaviors (or actions) from a combination of inputs from emotional, cognitive, and motor cortical areas. Recent anatomical evidence from primates demonstrates that the neuro-networks within basal ganglia pathways are in a position to move information across functional circuits. Two networks are: the striato-nigral-striatal network and the thalamo-cortical-thalamic network. Within each of these sets of connected structures, there are both reciprocal connections linking up regions associated with similar functions and non-reciprocal connections linking up regions that are associated with different cortical basal ganglia circuits. Each component of information (from limbic to motor outcome) sends both feedback connection, and also a feedforward connection, allowing the transfer of information. Information is channeled from limbic, to cognitive, to motor circuits. Action decision-making processes are thus influenced by motivation and cognitive inputs, allowing the animal to respond appropriate to environmental cues.},
author = {Haber, Suzanne N},
issn = {0891-0618},
journal = {Journal of Chemical Neuroanatomy},
keywords = {{Basal} {Ganglia,Humans,Mental} {Processes,Neural} {Pathways,Primates},{Animals}},
note = {{PMID:} 14729134
},
pages = {317--330},
title = {The primate basal ganglia: parallel and integrative networks},
url = {http://www.ncbi.nlm.nih.gov/pubmed/14729134},
volume = {26},
year = {2003}
}
@article{halpain_activation_1990,
abstract = {In the caudate-putamen the glutamatergic cortical input and the dopaminergic nigrostriatal input have opposite effects on the firing rate of striatal neurons. Although little is known of the biochemical mechanisms underlying this antagonism, one action of dopamine is to stimulate the cyclic {AMP-dependent} phosphorylation of {DARPP-32} (dopamine and {cAMP-regulated} phospho-protein, of relative molecular mass 32,000 {(32K].} This phosphorylation converts {DARPP-32} from an inactive molecule into a potent inhibitor of protein phosphatase-1. Here we show that activation of the {NMDA} {(N-methyl-D-aspartate)} subclass of glutamate receptors reverses the {cAMP-stimulated} phosphorylation of {DARPP-32} in striatal slices through {NMDA-induced} dephosphorylation of {DARPP-32.} Thus, the antagonistic effects of dopamine and glutamate on the excitability of striatal neurons are reflected in antagonistic effects of these neurotransmitters on the state of phosphorylation of {DARPP-32.} Our results indicate that stimulation of {NMDA} receptors leads to the activation of a neuronal protein phosphatase, presumably the calcium-dependent phosphatase calcineurin, and show, in an intact cell preparation, that signal transduction in the nervous system can be mediated by protein dephosphorylation.},
author = {Halpain, S and Girault, J A and Greengard, P},
doi = {10.1038/343369a0},
issn = {0028-0836},
journal = {Nature},
keywords = {{Aspartic} {Acid,Calcineurin,Calmodulin-Binding} {Proteins,Corpus} {Striatum,Cyclic} {AMP,Dibenzocycloheptenes,Dizocilpine} {Maleate,Dopamine} and {cAMP-Regulated} Phosphoprotein {32,Enzyme} {Activation,Forskolin,N-Methylaspartate,Nerve} Tissue {Proteins,Phosphates,Phosphoproteins,Phosphoprotein} {Phosphatases,Phosphorylation,Phosphothreonine,Protein} Phosphatase {1,Protein} {Kinases,Rats,Receptors,Valine},{Animals,} {N-Methyl-D-Aspartate,} {Neurotransmitter}},
note = {{PMID:} 2153935
},
pages = {369--372},
title = {Activation of {NMDA} receptors induces dephosphorylation of {DARPP-32} in rat striatal slices},
url = {http://www.ncbi.nlm.nih.gov/pubmed/2153935},
volume = {343},
year = {1990}
}
@article{hemmings_darpp-32dopamine-regulated_????,
abstract = {The neurotransmitter dopamine has been demonstrated by biochemical, histochemical and immunocytochemical techniques to be unevenly distributed in the mammalian central nervous system. {DARPP-32} (dopamine- and {cyclic-AMP-regulated} phosphoprotein of molecular weight 32,000) is a neuronal phosphoprotein that displays a regional distribution in the mammalian brain very similar to that of dopamine-containing nerve terminals, being highly concentrated in the basal ganglia. The state of phosphorylation of {DARPP-32} can be regulated by dopamine and by cyclic {AMP} in intact nerve cells, suggesting a role for this phosphoprotein in mediating certain of the effects of dopamine on dopaminoceptive cells. The observation that many of the physical and chemical properties of purified {DARPP-32} resemble those of phosphatase inhibitor-1 (inhibitor-1), a widely distributed inhibitor of protein phosphatase-1, suggests that {DARPP-32} might also function as a phosphatase inhibitor. We report here that {DARPP-32} inhibits protein phosphatase-1 at nanomolar concentrations. Moreover, like inhibitor-1, {DARPP-32} is effective as an inhibitor in its phosphorylated but not its dephosphorylated form. Thus, the basal ganglia of mammalian brain contain a region-specific neuronal phosphoprotein that is a protein phosphatase inhibitor.},
author = {Hemmings, H C and Greengard, P and Tung, H Y and Cohen, P},
issn = {0028-0836},
journal = {Nature},
keywords = {{Calcium,Calmodulin-Binding} {Proteins,Cattle,Caudate} {Nucleus,Cyclic} {AMP,Dopamine,Dopamine} and {cAMP-Regulated} Phosphoprotein {32,Nerve} Tissue {Proteins,Phosphoproteins,Phosphoprotein} {Phosphatases,Protein} Phosphatase 1,{Animals}},
note = {{PMID:} 6087160
},
pages = {503--505},
title = {{DARPP-32,} a dopamine-regulated neuronal phosphoprotein, is a potent inhibitor of protein phosphatase-1},
url = {http://www.ncbi.nlm.nih.gov/pubmed/6087160},
volume = {310}
}
@article{hemmings_darpp-32dopamine-_1984,
author = {Hemmings, H C and Nairn, A C and Aswad, D W and Greengard, P},
journal = {J. Neurosci.},
pages = {99--110},
title = {{DARPP-32,} a dopamine- and adenosine 3':5'-monophosphate-regulated phosphoprotein enriched in dopamine-innervated brain regions. {II.} Purification and characterization of the phosphoprotein from bovine caudate nucleus},
url = {http://www.jneurosci.org/cgi/content/abstract/4/1/99},
volume = {4},
year = {1984}
}
@article{hsieh_behavioral_2006,
author = {Hsieh, L T},
title = {Behavioral and {Event-Related} Potential Studies of {Item-Method} Directed Forgetting Effect},
year = {2006}
}
@article{huang_pp1_2001,
author = {Huang, H and Farley, J},
journal = {Journal of Neurophysiology},
pages = {1297--1311},
title = {{PP1} inhibitors depolarize Hermissenda photoreceptors and reduce K+ currents},
volume = {86},
year = {2001}
}
@article{janus_[beta]_2000,
author = {Janus, Christopher and Pearson, Jacqueline and McLaurin, JoAnne and Mathews, Paul M and Jiang, Ying and Schmidt, Stephen D and Chishti, M Azhar and Horne, Patrick and Heslin, Donna and French, Janet and Mount, Howard T J and Nixon, Ralph A and Mercken, Marc and Bergeron, Catherine and Fraser, Paul E and George-Hyslop, Peter St and Westaway, David},
doi = {10.1038/35050110},
issn = {0028-0836},
journal = {Nature},
pages = {979--982},
title = {A[beta] peptide immunization reduces behavioural impairment and plaques in a model of Alzheimer's disease},
url = {http://dx.doi.org/10.1038/35050110},
volume = {408},
year = {2000}
}
@article{joseph_long-term_1998,
abstract = {Recent research has indicated that increased vulnerability to oxidative stress may be the major factor involved in {CNS} functional declines in aging and age-related neurodegenerative diseases, and that antioxidants, e.g., vitamin E, may ameliorate or prevent these declines. Present studies examined whether long-term feeding of Fischer 344 rats, beginning when the rats were 6 months of age and continuing for 8 months, with diets supplemented with a fruit or vegetable extract identified as being high in antioxidant activity, could prevent the age-related induction of receptor-mediated signal transduction deficits that might have a behavioral component. Thus, the following parameters were examined: (1) oxotremorine-enhanced striatal dopamine release {(OX-K+-ERDA),} (2) cerebellar [beta] receptor augmentation of {GABA} responding, (3) striatal synaptosomal {45Ca2+} clearance, (4) carbachol-stimulated {GTPase} activity, and (5) Morris water maze performance. The rats were given control diets or those supplemented with strawberry extracts {(SE),} 9.5 gm/kg dried aqueous extract {(DAE),} spinach {(SPN} 6.4 gm/kg {DAE),} or vitamin E (500 {IU/kg).} Results indicated that {SPN-fed} rats demonstrated the greatest retardation of age-effects on all parameters except {GTPase} activity, on which {SE} had the greatest effect, whereas {SE} and vitamin E showed significant but equal protection against these age-induced deficits on the other parameters. For example, {OX-K+-ERDA} enhancement was four times greater in the {SPN} group than in controls. Thus, phytochemicals present in antioxidant-rich foods such as spinach may be beneficial in retarding functional age-related {CNS} and cognitive behavioral deficits and, perhaps, may have some benefit in neurodegenerative disease.},
author = {Joseph, J A and Shukitt-Hale, B and Denisova, N A and Prior, R L and Cao, G and Martin, A and Taglialatela, G and Bickford, P C},
journal = {J. Neurosci.},
pages = {8047--8055},
title = {{Long-Term} Dietary Strawberry, Spinach, or Vitamin E Supplementation Retards the Onset of {Age-Related} Neuronal {Signal-Transduction} and Cognitive Behavioral Deficits},
url = {http://www.jneurosci.org/cgi/content/abstract/18/19/8047},
volume = {18},
year = {1998}
}
@article{joslyn_directed_2005,
author = {Joslyn, S L and Oakes, M A},
journal = {{MEMORY} {AND} {COGNITION}},
pages = {577},
title = {Directed forgetting of autobiographical events},
volume = {33},
year = {2005}
}
@article{jouvenceau_different_2003,
author = {Jouvenceau, A and Billard, J M and Haditsch, U and Mansuy, I M and Dutar, P},
journal = {European Journal of Neuroscience},
pages = {1279--1285},
title = {Different phosphatase-dependent mechanisms mediate long-term depression and depotentiation of long-term potentiation in mouse hippocampal {CA1} area},
volume = {18},
year = {2003}
}
@article{jouvenceau_partial_2006,
author = {Jouvenceau, A and Hedou, G and Potier, B and Kollen, M and Dutar, P and Mansuy, I M},
journal = {European Journal of Neuroscience},
pages = {564--572},
title = {Partial inhibition of {PP1} alters bidirectional synaptic plasticity in the hippocampus},
volume = {24},
year = {2006}
}
@article{kraemer_adaptive_1997,
author = {Kraemer, P J and Golding, J M},
journal = {Psychonomic Bulletin and Review},
pages = {480--491},
title = {Adaptive forgetting in animals},
volume = {4},
year = {1997}
}
@article{kwon_characterization_1997,
abstract = {The catalytic subunit of {PP-1} {(PP-1C)} is potently inhibited {(IC50,} approximately 1 {nM)} by {DARPP-32} (dopamine- and {cAMP-regulated} phosphoprotein, M(r) 32,000), inhibitor-1, and inhibitor-2. The {NH2-terminal} 50 amino acid residues of {DARPP-32} and inhibitor-1 are similar, and phosphorylation of a common threonine residue {(Thr-34/Thr-35)} is necessary for inhibition of {PP-1C.} We have characterized further the interaction between {DARPP-32} and {PP-1C.} Using synthetic peptides derived from the {NH2-terminal} region of {DARPP-32,} residues 6-11, {RKKIQF,} have been shown to be required for inhibition of {PP-1C.} Peptides containing this motif were able to antagonize the inhibition of {PP-1C} by {phospho-DARPP-32} and phosphoinhibitor-1. The inhibition of {PP-1C} by inhibitor-2, but not by okadaic acid, microcystin, or calyculin A, was also attentuated by these antagonist peptides. These results together with results from other studies support a model in which two subdomains of {phospho-DARPP-32} interact with {PP-1C.} The region encompassing {phospho-Thr-34} appears to interact with the active site of the enzyme blocking enzyme activity. The region encompassing the {RKKIQF} motif binds to a domain of {PP-1C} removed from the active site. Amino acid sequence analysis indicates that basic and hydrophobic features of the {RKKIQF} motif are conserved in the binding domains of certain {PP-1C} targeting proteins, suggesting that interaction of inhibitor proteins and targeting proteins may be mutually exclusive.},
author = {Kwon, Y G and Huang, H B and Desdouits, F and Girault, J A and Greengard, P and Nairn, A C},
issn = {0027-8424},
journal = {Proceedings of the National Academy of Sciences of the United States of America},
keywords = {Amino Acid {Sequence,Animals,Dopamine} and {cAMP-Regulated} Phosphoprotein {32,Enzyme} {Inhibitors,Molecular} Sequence {Data,Nerve} Tissue {Proteins,Phosphoproteins,Phosphoprotein} {Phosphatases,Protein} Phosphatase {1,Protein} {Binding,Recombinant} Proteins},
note = {{PMID:} 9108011
},
pages = {3536--3541},
title = {Characterization of the interaction between {DARPP-32} and protein phosphatase 1 {(PP-1):} {DARPP-32} peptides antagonize the interaction of {PP-1} with binding proteins},
url = {http://www.ncbi.nlm.nih.gov/pubmed/9108011},
volume = {94},
year = {1997}
}
@article{lee_investigation_2007,
author = {Lee, S C},
title = {The investigation of item-method directed forgetting effect in the encoding and retrieval phases of recognition memory.},
year = {2007}
}
@article{lee_can_2007,
author = {Lee, Y},
journal = {Acta Psychologica},
title = {Can intentional forgetting reduce false memory? Effects of list-level and item-level forgetting},
year = {2007}
}
@article{macleod_concept_????,
author = {MacLeod, C M},
title = {The Concept of Inhibition}
}
@article{macleod_in_2003,
author = {MacLeod, C M and Dodd, M D and Sheard, E D and Wilson, D E and Bibi, U},
journal = {Psychology of learning and motivation},
pages = {163--215},
title = {In opposition to inhibition},
volume = {43},
year = {2003}
}
@article{mayerhofer_functional_1999,
abstract = {The catecholamines norepinephrine and dopamine {(DA)} are present in the human ovary; in particular, in follicular fluid. Norepinephrine activates ovarian alpha- and beta-adrenergic receptors and modulates ovarian steroidogenesis, but the significance of ovarian {DA} is unclear. We examined whether a {DA} receptor of the D1-subtype {(D1-R)} is present in human ovary and in cultured human granulosa luteal cells {(GC).} Using {RT-PCR,} we cloned complementary {DNAs} from adult human ovarian and {GC} messenger {RNAs,} which are identical to the human {D1-R} sequence. In ovarian sections, {D1-R} protein was identified (by immunohistochemistry) in granulosa cells of large antral follicles, cells of the corpus luteum, as well as in cultured {GC.} An immunoreactive band of approximately Mr 50,000 was found in cultured luteinized {GC} using the same antiserum in Western blots. The {D1-R} in these cells was functional, because {DA,} alone or in the presence of the beta-receptor antagonist propranolol, caused cellular contraction. The selective {D1-R} agonist {SKF-38393} induced a similar change in cytomorphology and increased the levels of media {cAMP.} {SKF-38393} failed, however, to significantly affect basal and {hCG-stimulated} progesterone release in vitro, indicating that the activation of the {D1-R} was not directly linked to synthesis of progesterone, the major steroid of human {GC.} Estradiol synthesis likewise was not affected. Using {RT-PCR} and immunohistochemistry, we found that {GC} express {DA-} and {cAMP-regulated} phosphoprotein of Mr 32,000 {(DARPP-32),} a protein typically associated with neurons bearing the {D1-R.} In cultured {GC,} {DA} and {SKF-38393} induced increased threonine-phosphorylation of {DARPP-32,} even in the presence of propranolol but not in the presence of {D1-R} antagonist {SCH-23390.} Taken together, the presence of {DA} and a functional {DA} receptor and {DARPP-32} indicate that a novel, physiological regulatory pathway involving {DA} exists in the human ovary.},
author = {Mayerhofer, A and Hemmings, H C and Snyder, G L and Greengard, P and Boddien, S and Berg, U and Brucker, C},
issn = {0021-972X},
journal = {The Journal of Clinical Endocrinology and Metabolism},
keywords = {{Base} {Sequence,Cells,Cyclic} {AMP,Dopamine} and {cAMP-Regulated} Phosphoprotein {32,Estradiol,Female,Granulosa} {Cells,Humans,Molecular} Sequence {Data,Nerve} Tissue {Proteins,Ovary,Phosphoproteins,Phosphorylation,Progesterone,RNA,Receptors,{Adult,} Dopamine {D1,} Messenger,} {Cultured}},
note = {{PMID:} 9920093
},
pages = {257--264},
title = {Functional dopamine-1 receptors and {DARPP-32} are expressed in human ovary and granulosa luteal cells in vitro},
url = {http://www.ncbi.nlm.nih.gov/pubmed/9920093},
volume = {84},
year = {1999}
}
@article{mcnab_common_2008,
author = {McNab, F and Leroux, G and STRAND, F and Thorell, L and Bergman, S and Klingberg, T},
journal = {Neuropsychologia},
pages = {2668--2682},
title = {Common and unique components of inhibition and working memory: An {fMRI,} within-subjects investigation},
volume = {46},
year = {2008}
}
@article{meyer-lindenberg_genetic_2007,
author = {Meyer-Lindenberg, Andreas and Straub, Richard E and Lipska, Barbara K and Verchinski, Beth A and Goldberg, Terry and Callicott, Joseph H and Egan, Michael F and Huffaker, Stephen S and Mattay, Venkata S and Kolachana, Bhaskar and Kleinman, Joel E and Weinberger, Daniel R},
doi = {10.1172/JCI30413},
journal = {Journal of Clinical Investigation},
note = {PMC1784004
},
pages = {672–682},
title = {Genetic evidence implicating {DARPP-32} in human frontostriatal structure, function, and cognition},
url = {http://www.pubmedcentral.nih.gov/articlerender.fcgi?artid=1784004},
volume = {117},
year = {2007}
}
@article{mitchell_directed_2002,
author = {Mitchell, J P and Macrae, C N and Schooler, J W and Rowe, A C and Milne, A B},
journal = {Levels of Processing 30 Years on: A Special Issue of Memory},
pages = {381--388},
title = {Directed remembering: Subliminal cues alter nonconscious memory strategies},
volume = {10},
year = {2002}
}
@article{morgandave_correction:[beta]_2001,
author = {MorganDave,  and M., DiamondDavid and E., GottschallPaul and E., UgenKenneth and DickeyChad,  and HardyJohn,  and DuffKaren,  and JantzenPaul,  and DiCarloGiovanni,  and WilcockDonna,  and ConnorKaren,  and HatcherJaime,  and HopeCaroline,  and GordonMarcia,  and W., ArendashGary},
doi = {10.1038/35088102},
issn = {0028-0836},
journal = {Nature},
pages = {660},
title = {correction: A[beta] peptide vaccination prevents memory loss in an animal model of Alzheimer's disease},
url = {http://dx.doi.org/10.1038/35088102},
volume = {412},
year = {2001}
}
@article{mller_directed_2005,
author = {Muller, U and Ullsperger, M and Hammerstein, E and Sachweh, S and Becker, T},
journal = {European archives of psychiatry and clinical neuroscience},
pages = {251--257},
title = {Directed forgetting in schizophrenia},
volume = {255},
year = {2005}
}
@article{munton_role_2004,
author = {Munton, R P and Vizi, S and Mansuy, I M},
journal = {{FEBS} letters},
pages = {121--128},
title = {The role of protein phosphatase-1 in the modulation of synaptic and structural plasticity},
volume = {567},
year = {2004}
}
@article{nowicka_reversed_2009,
author = {Nowicka, A and Jednoróg, K and Wypych, M and Marchewka, A},
journal = {International Journal of Psychophysiology},
pages = {97--102},
title = {Reversed old/new effect for intentionally forgotten words: An {ERP} study of directed forgetting},
volume = {71},
year = {2009}
}
@article{ouimet_distribution_1990,
abstract = {{DARPP-32,} a dopamine and cyclic {AMP-regulated} phosphoprotein, has been studied by light and electron microscopical immunocytochemistry in the rat caudatoputamen, globus pallidus and substantia nigra. In the caudatoputamen, {DARPP-32} was present in neurons of the medium-sized spiny type. Immunoreactivity for {DARPP-32} was present in dendritic spines, dendrites, perikaryal cytoplasm, most but not all nuclei, axons and a small number of axon terminals. Immunoreactive axon terminals in the caudatoputamen formed symmetrical synapses with immunolabeled dendritic shafts or somata. Neurons having indented nuclei were never immunoreactive. In the globus pallidus and substantia nigra pars reticulata, {DARPP-32} was present in myelinated and unmyelinated axons and in axon terminals. The labelled axon terminals in these regions formed symmetrical synaptic contacts on unlabelled dendritic shafts or on unlabelled somata. These data suggest that {DARPP-32} is present in striatal neurons of the medium-sized spiny type and that these {DARPP-32-immunoreactive} neurons form symmetrical synapses on target neurons in the globus pallidus and substantia nigra. The presence of {DARPP-32} in these striatal neurons and in their axon terminals suggests that {DARPP-32} mediates part of the response of medium-size spiny neurons in the striatum to dopamine D-1 receptor activation.},
author = {Ouimet, C C and Greengard, P},
issn = {0300-4864},
journal = {Journal of Neurocytology},
keywords = {{Basal} {Ganglia,Caudate} {Nucleus,Dopamine} and {cAMP-Regulated} Phosphoprotein {32,Globus} {Pallidus,Immunoenzyme} {Techniques,Male,Microscopy,Nerve} Tissue {Proteins,Neurons,Phosphoproteins,Putamen,Rats,Substantia} {Nigra,Synapses},{Animals,} Inbred {Strains,} {Electron}},
note = {{PMID:} 2191086
},
pages = {39--52},
title = {Distribution of {DARPP-32} in the basal ganglia: an electron microscopic study},
url = {http://www.ncbi.nlm.nih.gov/pubmed/2191086},
volume = {19},
year = {1990}
}
@article{ouimet_immunocytochemical_1992,
abstract = {The localization of {DARPP-32,} a dopamine and {cAMP-regulated} phosphoprotein, has been studied in monkey brain by immunocytochemistry. This study indicates that {DARPP-32} is enriched in neurons in regions receiving a dense dopamine input from the substantia nigra and ventral tegmental area. Thus, the majority of somata in the anterior olfactory area, nucleus accumbens, caudate nucleus, and putamen are immunoreactive for {DARPP-32.} In the caudate nucleus, immunoreactive spines receive asymmetric contacts from unlabeled axon terminals. Immunoreactive somata have diameters of 10-15 microns. In regions known to receive projections from these nuclei, immunoreactivity is confined to small puncta that represent axons and axon terminals. Regions in which immunoreactivity is present in puncta include the ventral pallidum, globus pallidus, and substantia nigra pars reticulata. Dopaminergic neurons themselves are not immunoreactive. Neurons containing moderate to weak immunoreactivity for {DARPP-32} are observed in portions of the cerebral cortex, particularly in the temporal cortex (layer {VI).} {DARPP-32-positive} neurons are also present in the cerebellum, in the medial habenula, and in portions of the bed nucleus of the stria terminalis and amygdaloid complex. {DARPP-32} immunoreactivity is also present in astrocytes in the subcortical white matter and in tanycytes in the arcuate nucleus and median eminence. {DARPP-32} may be an effective marker for dopaminoceptive neurons in which the actions of dopamine on the D-1 dopamine receptor are mediated through {cAMP} and its associated protein kinase.},
author = {Ouimet, C C and Lamantia, A S and Goldman-Rakic, P and Rakic, P and Greengard, P},
doi = {10.1002/cne.903230206},
issn = {0021-9967},
journal = {The Journal of Comparative Neurology},
keywords = {{Brain,Brain} {Chemistry,Cyclic} {AMP,Dopamine,Dopamine} and {cAMP-Regulated} Phosphoprotein {32,Immunohistochemistry,Macaca} {mulatta,Male,Microscopy,Nerve} Tissue {Proteins,Phosphoproteins,Receptors,Second} Messenger Systems,{Animals,} Dopamine {D1,} {Electron}},
note = {{PMID:} 1328330
},
pages = {209--218},
title = {Immunocytochemical localization of {DARPP-32,} a dopamine and {cyclic-AMP-regulated} phosphoprotein, in the primate brain},
url = {http://www.ncbi.nlm.nih.gov/pubmed/1328330},
volume = {323},
year = {1992}
}
@article{quartermain_amphetamine_1988,
author = {Quartermain, D and Judge, M E and Jung, H},
journal = {Physiology \& behavior},
pages = {239--241},
title = {Amphetamine enhances retrieval following diverse sources of forgetting},
volume = {43},
year = {1988}
}
@article{sahakyan_can_2003,
author = {Sahakyan, L and Delaney, P F},
journal = {Journal of Memory and Language},
pages = {195--206},
title = {Can encoding differences explain the benefits of directed forgetting in the list method paradigm?},
volume = {48},
year = {2003}
}
@article{silva_cognitive_2002,
author = {Silva, A J and Josselyn, S A},
journal = {Nature a-z index},
pages = {929--930},
title = {Cognitive {neuroscienceThe} molecules of forgetfulness},
volume = {418},
year = {2002}
}
@article{starns_episodic_2004,
author = {Starns, J J and Hicks, J L},
journal = {Memory and Cognition},
pages = {602--609},
title = {Episodic generation can cause semantic forgetting: Retrieval-induced forgetting of false memories},
volume = {32},
year = {2004}
}
@article{storm_short_????,
author = {Storm, B C and Bjork, E L and Bjork, R A},
title = {Short article When intended remembering leads to unintended forgetting}
}
@article{swerdlow_neural_2001,
abstract = {{RATIONALE:} Sensorimotor gating of the startle reflex can be assessed across species, using similar stimuli to elicit similar responses. Prepulse inhibition {(PPI),} a measure of sensorimotor gating, is reduced in patients with some neuropsychiatric disorders, and in rats after manipulations of limbic cortex, striatum, pallidum or pontine tegmentum {("CSPP"} circuitry). {OBJECTIVE:} To review the current knowledge of the neural circuit regulation of {PPI} in rats, and to anticipate the future challenges facing this line of inquiry. {METHODS:} The published literature was reviewed and critically evaluated. {RESULTS:} Limbic {CSPP} circuitry has been studied in rats to reveal the neurochemical and neuroanatomical substrates regulating {PPI} at a high level of resolution. In translational cross-species research, this detailed circuit information is used as a "blueprint" to identify substrates that may lead to {PPI} deficits in psychiatrically disordered humans. Some human disorders with identifiable, localized lesions in {CSPP} circuitry may provide direct validation for the contribution of {CSPP} circuitry to this cross-species model. The rapid collection of experimental data supporting this cross-species {PPI} circuit "blueprint" has supported continuing advances in the development of theoretical models for understanding how this circuitry normally functions to regulate {PPI.} Such models are needed for building a conceptual framework for understanding the role of this circuitry in the regulation of sensorimotor gating in normal humans, and in the relative loss of sensorimotor gating, and the resulting clinical consequences, in individuals with particular neuropsychiatric disorders. {CONCLUSIONS:} Our understanding of the neural regulation of {PPI} has increased tremendously over the past 15 years. Progress has come in "broad strokes", and a number of important details and complex questions remain to be addressed. It is anticipated that this is a "work in progress", and that the precise models for the neural regulation of {PPI} will evolve substantially in the coming years.},
author = {Swerdlow, N R and Geyer, M A and Braff, D L},
issn = {0033-3158},
journal = {Psychopharmacology},
keywords = {{Brain,Nerve} {Net,Rats,Startle} Reaction,{Animals}},
note = {{PMID:} 11549223
},
pages = {194--215},
title = {Neural circuit regulation of prepulse inhibition of startle in the rat: current knowledge and future challenges},
url = {http://www.ncbi.nlm.nih.gov/pubmed/11549223},
volume = {156},
year = {2001}
}
@article{toni_ltp_1999,
author = {Toni, N and Buchs, P.-A. and Nikonenko, I and Bron, C R and Muller, D},
doi = {10.1038/46574},
issn = {0028-0836},
journal = {Nature},
pages = {421--425},
title = {{LTP} promotes formation of multiple spine synapses between a single axon terminal and a dendrite},
url = {http://dx.doi.org/10.1038/46574},
volume = {402},
year = {1999}
}
@article{toni_ltp_1999,
author = {Toni, N and Buchs, P.-A. and Nikonenko, I and Bron, C R and Muller, D},
doi = {10.1038/46574},
issn = {0028-0836},
journal = {Nature},
pages = {421--425},
title = {{LTP} promotes formation of multiple spine synapses between a single axon terminal and a dendrite},
url = {http://dx.doi.org/10.1038/46574},
volume = {402},
year = {1999}
}
@article{torii_inhibitor_1995,
author = {Torii, N and Kamishita, T and Otsu, Y and Tsumoto, T},
journal = {Neuroscience Letters},
pages = {1--4},
title = {An inhibitor for calcineurin, {FK506,} blocks induction of long-term depression in rat visual cortex},
volume = {185},
year = {1995}
}
@article{valjent_regulation_2005,
abstract = {Many drugs of abuse exert their addictive effects by increasing extracellular dopamine in the nucleus accumbens, where they likely alter the plasticity of corticostriatal glutamatergic transmission. This mechanism implies key molecular alterations in neurons in which both dopamine and glutamate inputs are activated. Extracellular signal-regulated kinase {(ERK),} an enzyme important for long-term synaptic plasticity, is a good candidate for playing such a role. Here, we show in mouse that d-amphetamine activates {ERK} in a subset of medium-size spiny neurons of the dorsal striatum and nucleus accumbens, through the combined action of glutamate {NMDA} and D1-dopamine receptors. Activation of {ERK} by d-amphetamine or by widely abused drugs, including cocaine, nicotine, morphine, and Delta(9)-tetrahydrocannabinol was absent in mice lacking dopamine- and {cAMP-regulated} phosphoprotein of M(r) 32,000 {(DARPP-32).} The effects of d-amphetamine or cocaine on {ERK} activation in the striatum, but not in the prefrontal cortex, were prevented by point mutation of Thr-34, a {DARPP-32} residue specifically involved in protein phosphatase-1 inhibition. Regulation by {DARPP-32} occurred both upstream of {ERK} and at the level of striatal-enriched tyrosine phosphatase {(STEP).} Blockade of the {ERK} pathway or mutation of {DARPP-32} altered locomotor sensitization induced by a single injection of psychostimulants, demonstrating the functional relevance of this regulation. Thus, activation of {ERK,} by a multilevel protein phosphatase-controlled mechanism, functions as a detector of coincidence of dopamine and glutamate signals converging on medium-size striatal neurons and is critical for long-lasting effects of drugs of abuse.},
author = {Valjent, Emmanuel and Pascoli, Vincent and Svenningsson, Per and Paul, Surojit and Enslen, Herve and Corvol, Jean-Christophe and Stipanovich, Alexandre and Caboche, Jocelyne and Lombroso, Paul J and Nairn, Angus C and Greengard, Paul and Hervé, Denis and Girault, Jean-Antoine},
doi = {10.1073/pnas.0408305102},
issn = {0027-8424},
journal = {Proceedings of the National Academy of Sciences of the United States of America},
keywords = {{Cocaine,Corpus} {Striatum,Dextroamphetamine,Dopamine,Dopamine} and {cAMP-Regulated} Phosphoprotein {32,Enzyme} {Activation,Glutamic} {Acid,MAP} Kinase Signaling {System,Male,Mice,Mitogen-Activated} Protein {Kinases,Nerve} Tissue {Proteins,Phosphoproteins,Phosphorylation,Receptors,Signal} Transduction,{Animals,} Inbred {C57BL,} {N-Methyl-D-Aspartate}},
note = {{PMID:} 15608059
},
pages = {491--496},
title = {Regulation of a protein phosphatase cascade allows convergent dopamine and glutamate signals to activate {ERK} in the striatum},
url = {http://www.ncbi.nlm.nih.gov/pubmed/15608059},
volume = {102},
year = {2005}
}
@article{waddell_protein_2003,
author = {Waddell, S},
journal = {Trends in Neurosciences},
pages = {117--119},
title = {Protein phosphatase 1 and memory: practice makes {PP1} imperfect?},
volume = {26},
year = {2003}
}
@article{walaas_darpp-32dopamine-_1984,
author = {Walaas, S I and Greengard, P},
journal = {J. Neurosci.},
pages = {84--98},
title = {{DARPP-32,} a dopamine- and adenosine 3':5'-monophosphate-regulated phosphoprotein enriched in dopamine-innervated brain regions. I. Regional and cellular distribution in the rat brain},
url = {http://www.jneurosci.org/cgi/content/abstract/4/1/84},
volume = {4},
year = {1984}
}
@article{wessel_forgetting_2006,
author = {Wessel, I and Merckelbach, H},
journal = {Cognition \& Emotion},
pages = {129--137},
title = {Forgetting" murder" is not harder than forgetting" circle": Listwise-directed forgetting of emotional words},
volume = {20},
year = {2006}
}